Genetic Disposition for Global Maturity: An Explanation for Genetic Effects                 on Parental Report on ADHD

Steffensson, Birgitta; Larsson, Jan‐Olov; Fried, Ingegärd; El-Sayed, Eman; Rydelius, Per‐Anders; Lichtenstein, Paul

doi:10.1080/016502599383865

Cited by 25 publications

(28 citation statements)

References 36 publications

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“…Our results agree with theirs in that the increase in ADHD rates found by us was confined primarily to the school years and disappeared when parent‐rated symptoms of ADHD was studied in Cohort 2. Altogether, these findings are consistent with the idea that delayed maturation accounts for part of the aetiology of childhood ADHD (Steffensson et al., ) and its medication.…”

Section: Discussionsupporting

confidence: 90%

Relative immaturity and ADHD: findings from nationwide registers, parent‐ and self‐reports

Halldner

Tillander

Lundholm

et al. 2014

Child Psychology Psychiatry

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Relative immaturity compared to class mates might contribute to ADHD diagnosis and pharmacotherapy despite absence of parallel findings in reported ADHD symptom loads by relative immaturity. Increased clinical awareness of this phenomenon may be warranted to decrease risk for imprecise diagnostics and treatment. We speculate that flexibility regarding age at school start according to individual maturity could reduce developmentally inappropriate demands on children and improve the precision of ADHD diagnostic practice and pharmacological treatment.

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Section: Discussionsupporting

confidence: 90%

Relative immaturity and ADHD: findings from nationwide registers, parent‐ and self‐reports

Halldner

Tillander

Lundholm

et al. 2014

Child Psychology Psychiatry

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“…Different etiologies for aggression have been found for boys and girls in both childhood and adolescence (Vierikko et al, 2003) as well as in categorical ADHD (Eaves et al, 2000;Nadder et al, 2001;Steffensson et al, 1999;Rhee et al, 1999). In contrast, a similar pattern of genetic and environmental effects on boys and girls has been reported for children's and adolescents' maternal rated aggression (Eley et al, 1999) and parent-and teacher-rated ADHD (Nadder et al, 1998;Thapar et al, 2000).…”

mentioning

confidence: 93%

Genetic and Environmental Influences on the Relationship Between Aggression and Hyperactivity-impulsivity as Rated by Teachers and Parents

Vierikko¹,

Pulkkinen

Kaprio

et al. 2004

Twin Res

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T his study examined genetic and environmental contributions to the covariance between aggression and hyperactivity-impulsivity as rated by twins' teachers and parents. Sex-differences in these genetic and environmental contributions and rater bias/sibling interaction effects were of interest as well. Part of an ongoing nation-wide twin-family study of behavioral development and health habits, the sample consisted of 1636 Finnish twin pairs ascertained from five consecutive and complete twin birth cohorts. Data were collected at ages 11-12, using teacher and parental rating forms of the Multidimensional Peer Nomination Inventory. Bivariate analyses were performed using structural equation modeling allowing sex-limitation effects. Results show that, in addition to significant genetic and environmental influences specific to each behavior, aggression and hyperactivity-impulsivity share common genetic and environmental etiology. Results provide evidence that both genetic and environmental factors are important in creating the observed correlation between aggression and hyperactivity-impulsivity.Externalizing problems in children's behavior com

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“…These observations have given rise to the hypothesis that ADHD is related to a delay rather than a deviance of normal brain development (Kinsbourne, 1973; Steffensson et al, 1999; El-Sayed, 2002). …”

Section: Introductionmentioning

confidence: 99%

Maturational delay in ADHD: evidence from CPT

Berger

Slobodin

Aboud

et al. 2013

Front. Hum. Neurosci.

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While data from behavioral, neuropsychological, and brain studies suggested that Attention-Deficit/Hyperactivity Disorder (ADHD) is related to a developmental lag that reduces with age, other studies have proposed that ADHD represents a deviant brain function. The present study used a cross-sectional approach to examine whether ADHD children show a developmental delay in cognitive performance measured by continuous performance test (CPT). We thus, compared six age groups of ADHD children (N = 559) and their unaffected peers (N = 365), aged 6–11, in four parameters of MOXO-CPT performance: Attention, Timing, Hyperactivity and Impulsivity. Results have shown that despite improvement in CPT performance with age, ADHD children continued to demonstrate impaired performance as compared to controls. In most parameters, CPT performance of ADHD children matched that of 1–3 years younger normal controls, with a delay most prominent in older children. However, in the Hyperactivity parameter, ADHD children's performance resembled that of much younger healthy children, with almost no evidence for a developmental catch up. This study suggests that while some cognitive functions develop slower but normally, other functions (e.g., inhibitory control) show a different trajectory.

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Genetic Disposition for Global Maturity: An Explanation for Genetic Effects on Parental Report on ADHD

Cited by 25 publications

References 36 publications

Relative immaturity and ADHD: findings from nationwide registers, parent‐ and self‐reports

Relative immaturity and ADHD: findings from nationwide registers, parent‐ and self‐reports

Genetic and Environmental Influences on the Relationship Between Aggression and Hyperactivity-impulsivity as Rated by Teachers and Parents

Maturational delay in ADHD: evidence from CPT

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