Glaucoma due to Thrombosis of the Central Vein of the Retina

Prognosis for rubeosis iridis following central retinal vein occlusion.

“…However, none of the associated diseases alone showed a statistically significant difference from the cases of rubeosis with no associated disease. This seems to be contrary to the impression of others that rubeosis occurs more frequently in CRVO patients with primary glaucoma or systemic disease (Vannas, 1961;Kottow et al, 1976). Our observation that age was not significantly correlated with rubeosis iridis is also contrary to findings by Vannas (1961) and Kottow et al, (1976), but the increased number of elderly patients having rubeosis with CRVO in this study merely reflected the increased incidence of CRVO in the elderly.…”

“…This seems to be contrary to the impression of others that rubeosis occurs more frequently in CRVO patients with primary glaucoma or systemic disease (Vannas, 1961;Kottow et al, 1976). Our observation that age was not significantly correlated with rubeosis iridis is also contrary to findings by Vannas (1961) and Kottow et al, (1976), but the increased number of elderly patients having rubeosis with CRVO in this study merely reflected the increased incidence of CRVO in the elderly. This result, however, could be caused by an unrepresentative sample composed of the more severe CRVO patients referred to the Fluorescein Service.…”

“…Rubeosis iridis (RI) and neovascular glaucoma (NVG) are severe complications of central retinal vein occlusion (CRVO) which occur in 12% to 30% of all cases (Smith, 1961;Vannas, 1961;Laatikainen and Kohner, 1976). The stimulus for anterior segment neovascularisation is poorly understood but is thought to be related to severe retinal ischaemia (Smith, 1961;Ashton, 1961;Shilling and Kohner, 1976).…”

Electrophysiological evaluation and visual outcome in patients with central retinal vein occlusion, primary open‐angle glaucoma and neovascular glaucoma

“…Several clinical comparisons between CRVO and POAG have been published, and the prevalence of POAG in patients with CRVO has been reported to range from 6% to 69% (Bertelsen 1961; Vannas 1961; Dryden 1965; Soni & Woodhouse 1971; Luntz & Schenker 1980; Krakau et al. 1983; Frucht et al.…”

“…Several clinical comparisons between CRVO and POAG have been published, and the prevalence of POAG in patients with CRVO has been reported to range from 6% to 69% (Bertelsen 1961;Vannas 1961;Dryden 1965;Soni & Woodhouse 1971;Luntz & Schenker 1980;Krakau et al 1983;Frucht et al 1984;David et al 1988;Vadala et al 1997;Saatci et al 1999;Cursiefen et al 2001;Hayreh et al 2004). Further experimental investigations including histopathology have demonstrated the pathogenic similarity in these disorders, demonstrating endothelial proliferation in the retinal veins at the level of lamina cribrosa in both POAG and CRVO (Verhoeff 1913;Dryden 1965;Green et al 1981).…”