Gluteus Maximus Calcific Tendonosis

Singh, Jaspal; Yip, Kevin

doi:10.1097/phm.0000000000000190

Cited by 9 publications

(5 citation statements)

References 5 publications

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“…Full texts of the remaining 64 papers were obtained. A further 45 were excluded for not meeting the eligibility criteria, leaving 19 papers [[5], [6], [7], [8], [9], [10], [11], [12], [13], [14], [15], [16], [17], [18], [19], [20], [21], [22], [23]] in the review on which data extraction was performed. In total, 60 papers were excluded from the review for the following reasons: 15 non-English; 15 reviews of literature; 30 not tendonitis or enthesopathy.…”

Section: Resultsmentioning

confidence: 99%

Calcification of the linea aspera: A systematic narrative review

Low

Toms

2019

European Journal of Radiology Open

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Highlights Calcification at the linea aspera is a differential for hip/thigh pain. Imaging should commence with plain radiographs, and also include the distal femur. Smaller calcific deposits can be obscured on anteroposterior projection, necessitating additional views. Calcific tendo-enthesopathy is associated with osteolysis but no soft tissue mass.

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Section: Resultsmentioning

confidence: 99%

Calcification of the linea aspera: A systematic narrative review

Low

Toms

2019

European Journal of Radiology Open

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“…Other reports of gluteus maximus calcific tendinitis exist, but it is a relatively poorly known entity. [4][5][6][7][8][9] Because calcific tendinitis of the gluteus maximus is unusual and is associated with several nonspecific clinical and radiologic findings, it may present a diagnostic challenge. Clinically, the signs and symptoms may be suggestive of infection or inflammatory arthritis.…”

Section: Discussionmentioning

confidence: 99%

Calcific Tendinitis of the Gluteus Maximus in a Golfer

Williams

Stang²,

Fritz

et al. 2016

Orthopedics

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Calcific tendinitis is a relatively rare condition in which calcium is inappropriately deposited in tendons, resulting in a local inflammatory reaction that can cause severe symptoms in certain cases. The cause of this disease process is not completely understood, although repetitive microtrauma likely plays a role in its development. Although the disorder most often involves the rotator cuff, it can affect other structures throughout the body, such as the tendons about the ankle and hip-including the rectus femoris and gluteus maximus. Nonoperative management typically involves using an anti-inflammatory medication and activity modification and can be augmented with formal physical therapy and modalities. Although nonoperative management provides adequate relief for many patients, sometimes operative debridement of the calcific deposit with or without repair of the involved tendon is required. The authors report an unusual case of calcific tendinitis of the gluteus maximus insertion in a golfer. The patient had tried nonoperative treatment for approximately 2 years with no real relief, and a recent exacerbation of the pain was significantly delaying his return to sport. Although plain radiographs did not show abnormalities, magnetic resonance imaging showed a calcific deposit in the insertion of the gluteus maximus tendon. After discussing further treatment options with the patient, the decision was made to remove the deposit and repair the insertion. He recovered completely and was able to return to play. The frequency, pathogenesis, and treatment of this condition are discussed in this case report, as well as the possible link to golf in this patient. [Orthopedics.2016; 39(5):e997-e1000.].

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“…Physical therapy, a significant element in HADD management, works towards improving joint function, increasing range of motion, and strengthening muscles surrounding the affected joint [ 39 ]. NSAIDs, often employed to reduce inflammation and alleviate pain, function by inhibiting the production of certain inflammation-causing chemicals in the body [ 40 ].…”

Section: Treatment Of Haddmentioning

confidence: 99%

Hydroxyapatite Deposition Disease: A Comprehensive Review of Pathogenesis, Radiological Findings, and Treatment Strategies

Hegazi

2023

Diagnostics

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Hydroxyapatite deposition disease (HADD) represents a multifaceted condition characterized by the accumulation of hydroxyapatite crystals in soft tissues, leading to subsequent inflammation and discomfort. The intricate etiology of HADD is the subject of this comprehensive review, which encompasses an in-depth analysis of the four proposed pathogenic mechanisms and a deliberation on the predisposing factors that instigate the development of this disease. In order to provide a thorough understanding of the disease’s progression, this manuscript delineates the stages of HADD—those preceding calcification, occurring during calcification, and following calcification—in meticulous detail. This chronology forms the basis of a complete portrayal of the evolution of HADD. Moreover, this review encompasses an examination of the radiological findings associated with HADD, furnishing an extensive discourse on imaging characteristics. The potential of HADD to mimic other diseases, thereby posing diagnostic challenges, is also articulated. The discourse continues with an investigation of HADD’s differential diagnosis. This section furnishes a robust framework for distinguishing HADD from other conditions based on imaging results. To enrich the understanding of this diagnostic process, case studies illustrating real-world applications are provided. An overview of treatment modalities for HADD, including both conservative and interventional approaches, forms the concluding discussion. The pivotal role of imaging specialists in the diagnosis and management of HADD is emphasized, highlighting their vital contribution to image-guided procedures and disease monitoring.

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Gluteus Maximus Calcific Tendonosis

Cited by 9 publications

References 5 publications

Calcification of the linea aspera: A systematic narrative review

Calcification of the linea aspera: A systematic narrative review

Calcific Tendinitis of the Gluteus Maximus in a Golfer

Hydroxyapatite Deposition Disease: A Comprehensive Review of Pathogenesis, Radiological Findings, and Treatment Strategies

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