Glyphosate and glufosinate ammonium, herbicides commonly used on genetically modified crops, and their interaction with microplastics: Ecotoxicity in anuran tadpoles

Bioactive Compounds and Their Derivatives: An Insight into Prospective Phytotherapeutic Approach against Alzheimer’s Disease

“…The intrinsic antioxidant system regulates ROS balance in normal conditions [ 52 ]. However, there is a discrepancy between ROS generation and clearance in pathological situations, leading to high ROS levels [ 53 ]. Research findings indicated that brain OS may be an early event in AD and might affect disease development [ 54 , 55 ].…”

Glyphosate sensing in aqueous solutions by fluorescent zinc(ii) complexes of [9]aneN3-based receptors

“…Its appropriate use is considered safe, but concerns regarding its ecotoxicology are arising. 17 In recent years, different techniques have been developed for the monitoring and the detection of such important substrates in environmental matrices, including high performance liquid chromatography (HPLC), 18,19 gas chromatography-mass spectrometry (GC-MS), 20 ion chromatography (IC), 21 capillary electrophoresis (CE), 22 enzyme-linked immunosorbent assays (ELISA) 23 and optical 24 and electrochemical immuno-assays. [25][26][27] Most of these methods require expensive equipment and complex procedures, thus other methodologies have been developed to detect glyphosate, based on optical measurements, in particular fluorescence, that are highly sensitive, much simpler to operate, cost-effective, and rapid.…”

The integrated biomarker response in three anuran species larvae at sublethal concentrations of cypermethrin, chlorpyrifos, glyphosate, and glufosinate-ammonium

“…However, AChE of Scinax squalirostris tadpoles was inhibited after 48 h exposure to 1.26, 3.12, and 6.25 mg L -1 GLY. [62] Interestingly, in the same experiment Lajmanovich et al [62] found that GLU mixed with polyethylene microplastic particles (PEMPs) inhibited AChE of S. squalirostris when exposed to 1.26, 3.12 and 6.25 mg L -1 GLU during 48h, but GLU alone reduced AChE only at 6.25 mg L -1 . Farchi et al [63] linked elevated levels of neuronal AChE to muscle exhaustion by a combination of modest aberrations in synaptic transmission, muscle function, and muscle morphology.…”