Haemolytic Anaemia of Mechanical Origin after Open Heart Surgery

Sayed, Hoda Mohamed El; Dacie, J. V.; Handley, Dean A.; Lewis, Stanley M.; Cleland, W. P.

doi:10.1136/thx.16.4.356

Cited by 150 publications

(45 citation statements)

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“…1988; 8(4): [257][258][259][260] MeSH KEYWORDS: Anemia, hemolytic-infancy and childhood; Rheumatic heart disease THE INITIAL observation by Sayed et al 1 of intravascular erythrocyte destruction in a child whose ostium primum defect was repaired by Teflon patch was followed by numerous reports of hemolysis associated with intracardiac use of ball valve prosthesis 2 and Teflon cusps. 3 It has also been reported in malfunctioning mitral annuloplasty.…”

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confidence: 99%

Traumatic Hemolytic Anemia in Children with Rheumatic Heart Disease

1988

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Fifty children (5 to 15 years of age) with inactive compensated rheumatic heart disease were screened for traumatic hemolytic anemia. Eleven patients (22%) had normocytic hypochromic anemia. Their serum bilirubin and reticulocyte counts were significantly higher than those in the nonanemic group (P < 0.001); these levels suggest a hemolytic process. Direct Coombs' test was negative in all the patients studied. Haptoglobin level was significantly lower in the anemic group (P < 0.01). Only four cases (8%) showed hypohaptoglobinemia (serum haptoglobin less than 0.50 g/L), which denotes a mild intravascular hemolysis. The serum iron level was at the lower range of normal in these four patients, yet none of them had hemoglobinuria or hemosiderinuria.

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Traumatic Hemolytic Anemia in Children with Rheumatic Heart Disease

1988

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“…The patient had a positive direct antiglobulin test and circulating antibodies (anti-e and unidentified antibody), but no obvious signs of increased red-cell destruction could be detected. [4,7,8,11]. Recently [5] …”

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Transient Auto-Antibody Formation in a Case of Open Heart Surgery with no Signs of Increased Red-Cell Destruction

Hjelm¹,

Högman²,

Finnson³

et al. 1964

Vox Sanguinis

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“…SEVERE hemolytic anemia after surgical correction of congenital endocardial cushion defects is a rare clinical entity, first described by Sayed et al in 1961.1 Since then five more cases have been reported.2 4 All of these patients had an endocardial cushion defect with a cleft in the mitral valve. All defects were repaired with a Teflon patch, and the patients developed hemolytic anemia in the postoperative period.…”

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“…-4 Only one patient is reported to have demonstrated spontaneous remission although, after 8 months, splenomegaly and elevated reticulocyte count still persisted.2 Sayed's patient had an immediate cessation of hemolysis after a second operation at which the bare Teflon patch was covered with endocardium. 1 The remaining four patients were reoperated upon. Of the two patients reported by Sigler et The purpose of this paper is to describe the spontaneous remission of a hemolytic anemia that developed after repair of an ostium primum defect with a Teflon patch.…”

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confidence: 99%