2006
DOI: 10.1111/j.1399-6576.2006.01195.x
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Hemodynamic effect of sustained pulmonary hyperinflation in patients after cardiac surgery: open vs. closed chest

Abstract: Contrary to our hypothesis, sustained pulmonary hyperinflations with the chest open, i.e. before sternal closure, had similar negative effects on central hemodynamics as those performed with the chest closed, i.e. after sternal closure.

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Cited by 4 publications
(3 citation statements)
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“…50,63 The hemodynamic effects of recruitment maneuvers are observed even under open-chest conditions 64,65 and appear to decrease LV diastolic dimensions. 65 Under greatly exaggerated conditions of dynamic hyperinflation, complete cardiovascular collapse can result. 66 The selective delivery of oxygen to 1 lung results in alveolar hypoxia in the nonventilated lung.…”
Section: Olv: Effect On Cardiac Preload Afterload and Systolic Funcmentioning
confidence: 99%
See 1 more Smart Citation
“…50,63 The hemodynamic effects of recruitment maneuvers are observed even under open-chest conditions 64,65 and appear to decrease LV diastolic dimensions. 65 Under greatly exaggerated conditions of dynamic hyperinflation, complete cardiovascular collapse can result. 66 The selective delivery of oxygen to 1 lung results in alveolar hypoxia in the nonventilated lung.…”
Section: Olv: Effect On Cardiac Preload Afterload and Systolic Funcmentioning
confidence: 99%
“…62 More dramatic increases in lung volume and ITP associated with recruitment maneuvers 63,64 and higher levels of PEEP and CPAP during OLV are associated with a decrement in cardiac output. 50,63 The hemodynamic effects of recruitment maneuvers are observed even under open-chest conditions 64,65 and appear to decrease LV diastolic dimensions. 65 Under greatly exaggerated conditions of dynamic hyperinflation, complete cardiovascular collapse can result.…”
Section: Olv: Effect On Cardiac Preload Afterload and Systolic Functionmentioning
confidence: 99%
“…Вместе с тем, МА, как вариант ИВЛ с высоким положительным давлением в конце выдоха (ПДКВ), может вызывать разнонаправлен ные изменения основных детерминант доставки кис лорода (DO 2 ) -содержания последнего в артериаль ной крови (СаО 2 ) и сердечного выброса (СВ) [12]. Кардиодепрессивный эффект основного этапа МА, обеспечиваемой за счет повышения давления в дыха тельных путях, описан в ряде исследований, выпол ненных в ранние сроки после ИК [13,14]. При этом могут появляться признаки напряжения КТФ [14].…”
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