Hemodynamic evaluation for closing interatrial communication after fenestrated Fontan operation

Senzaki, Hideaki; C, Naito; Masutani, Satoshi; Nogaki, Mio; Ohono, Atsuko; Kobayashi, Jun; Sasaki, Nozomu; Asano, Haruhiko; Kyo, Shunei; Yokote, Yuji; Kobayashi, Toshiki

doi:10.1067/mtc.2001.111376

Cited by 8 publications

(2 citation statements)

References 4 publications

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“…Therefore, we will briefly introduce the method of assessing RV-PA interaction or RV-PA coupling. The ventricular pressure-volume relationship is the best way to demonstrate this [9][10][11]. An instantaneous plot of ventricular pressure and volume change yields a pressure-volume loop [12].…”

Section: Right Ventricular Function and Coupling To Pa Loadmentioning

confidence: 99%

Pathophysiology of Pulmonary Circulation in Congenital Heart Disease

Hashimoto

Kaneko

Nishida

et al. 2020

Molecular Mechanism of Congenital Heart Disease and Pulmonary Hypertension

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There are several types of abnormalities in the integrated physiology of pulmonary circulation in congenital heart disease. The main pathology of Eisenmenger syndrome involves a change in pulmonary resistance and is the most commonly observed pathophysiology in pulmonary hypertension. Other diseases also present with the main pathophysiological characteristic of reduced pulmonary compliance, such as tetralogy of Fallot and multiple peripheral pulmonary stenosis. In addition, the cavo-pulmonary connection has the unique feature of both pulmonary circulation and regulation. According to the differences in the pathophysiological features of pulmonary circulation, therapeutic approaches may considerably differ between diseases and conditions. Physiology-based clinical insights with regard to pulmonary circulation in congenital heart disease will be discussed in this chapter.

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Section: Right Ventricular Function and Coupling To Pa Loadmentioning

confidence: 99%

Pathophysiology of Pulmonary Circulation in Congenital Heart Disease

Hashimoto

Kaneko

Nishida

et al. 2020

Molecular Mechanism of Congenital Heart Disease and Pulmonary Hypertension

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“…After routine cardiac catheterization and before angioventriculography, ventricular P-A relations were constructed by simultaneously measuring ventricular chamber pressure and area both at rest and during preloads varied by transient inferior vena caval (IVC) balloon obstruction. Details of this procedure have previously been reported (10,12,13). Briefly, ventricular pressure was measured with a high-fidelity pressure transducer mounted on a 0.014-inch diameter guidewire (Radi Medical Systems, Inc., Wilmington, Massachusetts) in a 4-or 5-F pigtail catheter.…”

Section: Patientsmentioning

confidence: 99%

Cardiac Rest and Reserve Function in Patients With Fontan Circulation

Senzaki

Masutani

Ishido

et al. 2006

Journal of the American College of Cardiology

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155

104

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Because normal ventricular-vascular interaction and augmentation of cardiac performance during increased HR and adrenergic stimulation are important for maintaining cardiac output and exercise capacity, the present results may have important implications for the mechanisms underlying adverse outcome after Fontan surgery. Thus, improvement of long-term prognosis of patients after Fontan surgery requires the development of medical interventions that can overcome such limitations inherent in Fontan circulation.

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Considerations in Catheter Interventions

Cheung¹,

Benson²

2005

Ventricular Function and Blood Flow in Congenital Heart Disease

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Hemodynamic evaluation for closing interatrial communication after fenestrated Fontan operation

Cited by 8 publications

References 4 publications

Pathophysiology of Pulmonary Circulation in Congenital Heart Disease

Pathophysiology of Pulmonary Circulation in Congenital Heart Disease

Cardiac Rest and Reserve Function in Patients With Fontan Circulation

Considerations in Catheter Interventions

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