2008
DOI: 10.1016/j.jhep.2007.08.008
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Hepatitis C virus core protein impairs in vitro priming of specific T cell responses by dendritic cells and hepatocytes

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Cited by 45 publications
(38 citation statements)
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“…Likewise, expression of CCR7, a chemokine receptor that drives DC migration to secondary lymphoid organs, failed to be induced in A.BY/SnJ mice. In fact, DC maturation defects have been demonstrated in other viral infection models, which are prone to chronic disease like HIV [21] and HCV infection [22], emphasizing the importance of this finding in disease progression to ongoing myocarditis in CVB3-infected A.BY/SnJ mice. Notably, expression of MHC-I and MHC class II (MHC-II) molecules was affected only to a minor extent in both CVB3-challenged C57BL/6 and A.BY/SnJ mice (Fig.…”
Section: Maturation/activation In Cvb3-myocarditismentioning
confidence: 97%
“…Likewise, expression of CCR7, a chemokine receptor that drives DC migration to secondary lymphoid organs, failed to be induced in A.BY/SnJ mice. In fact, DC maturation defects have been demonstrated in other viral infection models, which are prone to chronic disease like HIV [21] and HCV infection [22], emphasizing the importance of this finding in disease progression to ongoing myocarditis in CVB3-infected A.BY/SnJ mice. Notably, expression of MHC-I and MHC class II (MHC-II) molecules was affected only to a minor extent in both CVB3-challenged C57BL/6 and A.BY/SnJ mice (Fig.…”
Section: Maturation/activation In Cvb3-myocarditismentioning
confidence: 97%
“…One of the most important biological features which should be taken into account is the pore size of the bone scaffolds (Annabi et al, 2009;Cicuéndez et al, 2012;Sondergaard et al, 2012;Zimmermann et al, 2008). The appropriate range of porosity for bone regeneration was determined through experimental investigations of biomaterials (Yang et al, 2001).…”
Section: Fabrication Of Scaffoldmentioning
confidence: 99%
“…Essential data pertaining to the function of NF-kB, are discussed below in relation to HCV-and HBV-induced chronic hepatitis. These viruses perpetuate liver inflammation, inhibit hepatocyte apoptosis and also inhibit the function of antigen-presenting DC (Thumann et al, 2008;Zimmermann et al, 2008). The latter effect has a deleterious impact on the adaptive immune response to viral peptides.…”
Section: Nf-jb In Chronic Hepatitismentioning
confidence: 99%