Hyperacute measurement of brain-tissue oxygen, carbon dioxide, pH, and intracranial pressure before, during, and after cerebral angiography in patients with aneurysmatic subarachnoid hemorrhage in poor condition

Anemia is Associated with Metabolic Distress and Brain Tissue Hypoxia After Subarachnoid Hemorrhage

“…It has been demonstrated that blood transfusions increase hemoglobin levels and have a direct impact on brain tissue oxygen tension (PbtO2) [9][10][11]. Observational studies have also shown that derangements of PbtO2 and markers of cerebral metabolism, such as glucose, lactate, and pyruvate, are associated with ischemia and poor outcome in patients with SAH [12][13][14][15][16][17][18][19][20][21][22]. Recent data suggest a relationship between anemia and deranged cerebral metabolism in the aftermath of SAH [23].…”

Reviewing the reliability, effectiveness and applications of Licox in traumatic brain injury

“…The majority of publications regarding the use of Licox analyses data from patients with severe TBI, with or without multiple extracranial injuries (Van Santbrink et al , 1996, 2003; Le Roux et al , 1997; Steel et al , 2005; Narotam et al , 2006; Bhatia and Gupta 2007; Longhi et al , 2007). The second largest group consists of patients with aneurysmal subarachnoid haemorrhage (Jaeger et al , 2003, 2005; Soehle et al , 2003; Nievas et al , 2005). Few studies have investigated the use of brain tissue oxygen monitoring in non‐traumatic subarachnoid haemorrhage (Hemphill et al , 2005), intraoperatively during aneurysm and cerebral arteriovenous malformation surgery (Jodicke et al , 2003) and during cerebral angiography (Nievas et al , 2005).…”

“…The second largest group consists of patients with aneurysmal subarachnoid haemorrhage (Jaeger et al , 2003, 2005; Soehle et al , 2003; Nievas et al , 2005). Few studies have investigated the use of brain tissue oxygen monitoring in non‐traumatic subarachnoid haemorrhage (Hemphill et al , 2005), intraoperatively during aneurysm and cerebral arteriovenous malformation surgery (Jodicke et al , 2003) and during cerebral angiography (Nievas et al , 2005). Major paediatric trauma cases have been studied with similar results in adults (Narotam et al , 2006).…”

Monitoring and Detection of Vasospasm II: EEG and Invasive Monitoring

“…A subsequent study with 11 patients published in 2001 found a strong correlation in [11] demonstrated the first direct correlation between PtiO 2 and the development of ischemic events in 32 patients. Two later studies correlated PtiO 2 values with angiographic vasospasm (N = 14) [12], and patient outcome (N = 42) [13]. In 2005, Jaeger et al [14] in Germany demonstrated that the level of PtiO 2 seems to be predominately determined by regional CBF, since changes in PtiO 2 were correlated in 90% of episodes to simultaneous changes of CBF.…”