Hypereosinophilic Syndrome With Eosinophilic Gastritis

Nayak, Vignesh Hebri; Engin, Nesrin Yurttutan; Burns, James; Ameta, Priyanka

doi:10.1177/2333794x17705239

Cited by 3 publications

(1 citation statement)

References 14 publications

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“…Sang-Hoon Lee 1 , Seung-Joo Nam 1 , Sung Joon Lee 1 , Chang Don Kang 1 , Dae Hee Choi 1 , Jang Won Choi 1 , and SeungKoo Lee 2 Departments of 1 Internal Medicine, 2 Anatomic Pathology, Kangwon National University School of Medicine, Chuncheon, Korea Eosinophilic gastritis is characterized by eosinophilic infiltration of the gastrointestinal tract, which is associated with abdominal pain, diarrhea, nausea, and vomiting. The possible etiologies of secondary eosinophilic gastritis, including drug reactions, parasitic infestation, and malignancy, must be evaluated.…”

Section: Praziquantel Treatment Of Eosinophilic Gastritis Suspected Tmentioning

confidence: 99%

Praziquantel Treatment of Eosinophilic Gastritis Suspected to Be Due to Cerebral Sparganosis

Lee¹,

Nam²,

Lee³

et al. 2018

Korean J Med

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Eosinophilic gastritis is characterized by eosinophilic infiltration of the gastrointestinal tract, which is associated with abdominal pain, diarrhea, nausea, and vomiting. The possible etiologies of secondary eosinophilic gastritis, including drug reactions, parasitic infestation, and malignancy, must be evaluated. Herein we report the case of a 65-year-old North Korean defector who presented with nausea and vomiting for 1 year. Secondary (reactive) eosinophilic gastritis caused by cerebral sparganosis was suspected after a workup that included brain magnetic resonance imaging, cerebrospinal fluid tapping, and gastric mucosal biopsy. The patient showed dramatic clinical improvement with high-dose praziquantel treatment. Even though secondary (reactive) eosinophilic gastritis caused by parasites is very rare, this case shows the importance of considering parasitic infection in eosinophilic gastritis. (Korean J Med 2018;93:473-476) 서 론 호산구성 위염(eosinophilic gastritis)은 위장관에 호산구가 침윤하여 염증을 일으키는 질환으로 오심, 구토, 복통, 설사, 폐쇄 등의 증상을 보인다. 호산구성 위염은 특별한 원인 없 이 위장에 호산구의 침윤이 발생하기도 하나, 이차적으로 호 산구의 침윤이 발생할 수도 있으며 기생충 감염, 약물 반응, 알레르기, 악성종양, 자가면역 질환 등에 의하여 발생할 수 있다[1]. 저자들은 호산구성 위염으로 내원한 환자에게서 병 력과 검사를 바탕으로 뇌 스파르가눔증(cerebral sparganosis) 이 의심되어 고용량 프라지콴텔(praziquantel)을 투여한 후 위 장관 증상의 소실과 호산구성 위염의 내시경적, 조직학적 호 전을 경험하여 문헌고찰과 함께 보고하고자 한다.

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Section: Praziquantel Treatment Of Eosinophilic Gastritis Suspected Tmentioning

confidence: 99%

Praziquantel Treatment of Eosinophilic Gastritis Suspected to Be Due to Cerebral Sparganosis

Lee¹,

Nam²,

Lee³

et al. 2018

Korean J Med

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The Cloaked Intruder: An Unexpected Case of Eosinophilic Cholangitis

et al. 2019

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Often, when biliary strictures are evaluated, malignancy is high on the differential. However, it is important to consider benign and malignant causes because approximately 10% of biliary strictures are benign. Eosinophilic cholangitis is an extremely rare benign disorder of the biliary tract caused by fibrosis and stricture from eosinophilic infiltration. The etiology of the disease remains unclear, and often, patients present with obstructive jaundice. We present a young man who presented with obstructive jaundice and abdominal pain, with a biliary stricture and peripheral eosinophilia. The final surgical pathology revealed eosinophilic cholangitis.

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An Immunohistochemical Assessment of Ki-67 and P53 Over-expression in Helicobacter pylori-positive Gastritis

Tawfeeq

Hamdi²,

Alazzo

2022

Open Access Maced J Med Sci

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AIM: Helicobacter pylori infection of the stomach is a common disease and the resulting changes from it are many and deserve to be in the focus of researchers’ attention, to assess the expression of mutant P53 protein and Ki-67 markers in patients with gastritis secondary to H. pylori. METHODS: Thirty samples with positive H. pylori gastritis were included in a retrospective study in Mosul/Iraq. The histological parameters were assessed using the Sydney system, then, the expression of Ki-67 and P53 expression was studied by immunohistochemical methods. The significance level was appointed at (0.05). RESULTS: Ki-67 and P53 expression were found in 83.3% of the total cases. The study results show that 92% of positive Ki-67 and P53 cases had chronic inflammatory cell infiltration, polymorph nuclear cells infiltration, and atrophy. Whereas 96% of positive Ki-67 cases had no metaplasia, 92% of the positive P53 cases had no metaplasia. The results also showed that only 16% of the positive Ki-67 cases had dysplastic changes, and 24% of the positive cases of P53 cases were showed dysplasia. whenever P53 was negative; there is neither metaplasia nor dysplasia in the tissue, this does not apply to Ki-67 negative cases. CONCLUSIONS: Ki-67 and P53 expressions increase with chronicity of Helicobacter pylori-positive gastritis. P53 expression is amplified when atrophy is present in these samples.

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Hypereosinophilic Syndrome With Eosinophilic Gastritis

Cited by 3 publications

References 14 publications

Praziquantel Treatment of Eosinophilic Gastritis Suspected to Be Due to Cerebral Sparganosis

Praziquantel Treatment of Eosinophilic Gastritis Suspected to Be Due to Cerebral Sparganosis

The Cloaked Intruder: An Unexpected Case of Eosinophilic Cholangitis

An Immunohistochemical Assessment of Ki-67 and P53 Over-expression in Helicobacter pylori-positive Gastritis

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