Hypokalaemic rhabdomyolysis

Kishore, Bhuvan; Thurlow, Vanessa; Kessel, B.

doi:10.1258/000456307780480882

Cited by 17 publications

(12 citation statements)

References 11 publications

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“…Another report demonstrated that 1 of 3 patients with acquired hypokalemic paralysis presented with a mild increase in CK levels after admission [4]. Other case reports demonstrated increased CK levels after admission [12,13]. Increased CK levels in acquired hypokalemic paralysis have been associated with muscle necrosis [11].…”

Section: Discussionmentioning

confidence: 99%

Clinical features and recovery patterns of acquired non-thyrotoxic hypokalemic paralysis

Hiraga

Kamitsukasa

Kojima

et al. 2012

Journal of the Neurological Sciences

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Section: Discussionmentioning

confidence: 99%

Clinical features and recovery patterns of acquired non-thyrotoxic hypokalemic paralysis

Hiraga

Kamitsukasa

Kojima

et al. 2012

Journal of the Neurological Sciences

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“…In our patient, ARF rapidly developed, probably due to rhabdomyolysis accompanying acidosis and dehydration caused by severe diarrhea. The reported causes of hypokalemic rhabdomyolysis include renal tubular acidosis, Bartter syndrome, diuretics, laxative, Conn's syndrome, etc (12,13). The first case of hypokalemic rhabdomyolysis caused by CD was reported in 1982; since then, six additional cases have been reported in which CD started with rhabdomyolysis induced by hypokalemia (2)(3)(4)(5).…”

Section: Discussionmentioning

confidence: 99%

Hypokalemic Rhabdomyolysis Induced Acute Renal Failure As A Presentation Of Coeliac Disease

Sarı¹,

Güneş²,

Sarıkaya³

et al. 2010

TUTFD

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Adult coeliac disease commonly presents without classical symptoms as chronic diarrhea and weight loss. We describe the case of a 31-year-old woman with persistent life-threatening hypokalemia, acute renal failure, and acute quadriplegia due to diarrhea that had continued for one month. Although there are cases of coeliac disease diagnosed with hypokalemic rhabdomyolysis in the literature, none of the cases developed acute renal failure. This is the first case in the literature diagnosed with acute renal failure due to hypokalemic rhabdomyolysis as a presentation of coeliac disease. In acute renal failure cases that present with hypokalemic rhabdomyolysis due to severe diarrhea, coeliac disease should be considered as a differential diagnosis despite the negative antigliadin IgA antibody.

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“…2 Conditions that promote potassium loss in critically ill patients include the response to stress, 2 treatment with mechanical ventilation, 3 hypertension, diarrhea, alcohol withdrawal, 4 and gastric draining or gavage.…”

Section: Purpose and Significance Of The Studymentioning

confidence: 99%

“…2 In instances of marked potassium loss, paralysis, rhabdomyolysis, myocardial infarction, and sudden death can occur. 1,4,14 Hypokalemia has also been associated with difficulty in weaning from mechanical ventilation 2,3 and subsequently can cause an increase in length of stay in the intensive care unit (ICU) and the hospital.…”

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confidence: 99%

Preventing Hypokalemia in Critically Ill Patients

Scotto

Fridline

Menhart

et al. 2014

American Journal of Critical Care

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Background Many therapies used in critical care cause potassium depletion. Current practice relies on potassium replacement protocols after a patient becomes hypokalemic. Potassium bolus therapy creates risk for patients, is costly, and increases nurses' workload. Objectives To determine if administering potassium preemptively in maintenance intravenous fluid would prevent episodes of hypokalemia and reduce the need for potassium boluses. Methods Medical records of 267 patients with normal potassium and creatinine levels at admission who did not receive total parenteral nutrition were reviewed. The 156 patients who met the study criteria were categorized by group: those who received potassium via maintenance intravenous fluid (treatment; n = 76) and those who did not (control; n = 80). The treatment group had potassium chloride or acetate added to intravenous fluid delivered at 36 to 72 mmol/d. Results The 2 groups did not differ significantly in age, race, sex, or admitting diagnosis. Type of diagnosis, length of stay, and potassium and creatinine levels at admission did not affect the number of potassium boluses for either group. The patients given maintenance potassium preemptively received significantly fewer (P < .001) potassium boluses (0.8) than did the control group (2.73), for a mean savings of $231 per patient for the treatment group. Conclusions Patients with normal potassium and creatinine levels at admission benefitted from a maintenance intravenous dose of potassium of 72 to 144 mmol/L per day. Compared with control patients, patients receiving this dose avoided detrimental hypokalemic events, had fewer invasive procedures and lower costs, and required less nursing care.

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Hypokalaemic rhabdomyolysis

Abstract: Addresses

Cited by 17 publications

References 11 publications

Clinical features and recovery patterns of acquired non-thyrotoxic hypokalemic paralysis

Clinical features and recovery patterns of acquired non-thyrotoxic hypokalemic paralysis

Hypokalemic Rhabdomyolysis Induced Acute Renal Failure As A Presentation Of Coeliac Disease

Preventing Hypokalemia in Critically Ill Patients

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