Incidence of cancer among workers producing calcium carbide.

Kjuus, Helge; Andersen, A; Langård, Sverre

doi:10.1136/oem.43.4.237

Cited by 20 publications

(16 citation statements)

References 9 publications

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“…Although fire fighters are exposed to complex inixtures of potentially carcinogenic substances, among these, PAH have established carcinogenic properties. Thus PAH may be an inlportant factor in the development of prostate cancer, although their exact etiologic role remains unclear (23,(38)(39)(40). Liou et al (41) inonitored PAH-DNA adducts, as a measure of potential carcinogenic damage associated with exposure to PAH.…”

Section: Biological Plausibilitymentioning

confidence: 99%

Association between diesel exposure at work and prostate cancer

Seidler¹,

Heiskel²,

Bickeböller³

et al. 1998

Scand J Work Environ Health

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Seidler A, Heiskel H, Bickeboller R, Elsner G. Association between diesel exposure at work and prostate cancer.Scand J Work Environ Health 1 998;24(6):486-494. ObjectivesThe possible etiologic relevance of occupational factors such as cadmium, cutting oils, diesel fuel and fumes, herbicides, polycyclic aromatic hydrocarbons (PAH), polychlorinated biphenyls, soot, tar, mineral oil, and solvents to prostate cancer was studied. Methods A case-referent study design was used to recruit 192 subjects with histologically confirmed prostate cancer and 210 referents who had prostate cancer histologically excluded either in orie of two urologic practices (Hamburg and Frankfurt) or in the urological policliilic of the Frankfurt University. Data were gathered with a self-administered questionnaire and analyzed using logistic regression to control for age, region, and cigarette smoking. A job-exposure matrix was used for assigning exposure. For the calculation of dose-years, the duration of contact with specific substa~lces was weighted by the intensity and probability of exposure according to a jobexposure matrix. Results The analysis of dose-years yielded a statistically significant association between occupational exposure to diesel fuel or fumes and prostate cancer (odds ratio 3.7,95% confidence interval 1.4-9.8, for subjects exposed to more than 25 dose-years in a co~npa~ison with subjects never exposed). For the other substances, no statistically significant differences in exposure were found between the cases and referents. When only jobs with a high exposurc probability were used to classify the participants as exposed, only exposure to PAH was significantly associated with prostate cancer. Conclusions In keeping with results from other studies, this study provides further evidence that exposure to diesel fuel or furnes -possibly mediated through PAH -may be associated with the developmeat of prostate cancer.

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Section: Biological Plausibilitymentioning

confidence: 99%

Association between diesel exposure at work and prostate cancer

Seidler¹,

Heiskel²,

Bickeböller³

et al. 1998

Scand J Work Environ Health

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“…Effects may include dizziness, frequent thirst, irritation in mouth and nose, weakness, permanent skin damage, difficulty in swallowing, vomiting, and skin ulcer, among others. The United States Department of Health [4] states that higher exposures may even cause pulmonary edema, and Kjuus et al [5] observed that chemicals including CaC 2 and acetylene gas also show other adverse effects including memory loss, neurological system failure, and cerebral edema. Other effects especially from contaminated foods include colonic lung cancer, quick-buck syndrome, DNA, RNA and hematological changes [6] [7], as well as proliferation of bacteria, fungi and viruses which can cause diarrhea, peptic ulcer and other human diseases.…”

Section: Introductionmentioning

confidence: 99%

Calcium Carbide-Induced Haematological Alterations In The Albino Mice- Mus musculus

Ogbuagu¹,

Ujowundu²,

Izunobi³

2016

IOSR

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“…Thirty-two deaths from lung cancer gave an SMR of 0.97 (95% CI, 0.67-1.37). Five deaths were from urinary bladder cancer (SMR, 1.08; 95% CI, 0.35-2.53) and five were from laryngeal cancer (SMR, 1.32;95% CI, Calcium carbide production Kjuus et al (1986) reported on the incidence of cancer in a cohort of 790 male Norwegian calcium carbide production workers. All workers who had been employed for at least 18 months between 1953 and 1970 were followed for cancer incidence and mortality until 1983.…”

Section: 17mentioning

confidence: 99%

“…Calcium carbide production Kjuus et al (1986) reported on the incidence of cancer in a cohort of 790 male Norwegian calcium carbide production workers. All workers who had been employed for at least 18 months between 1953 and 1970 were followed for cancer incidence and mortality until 1983.…”

mentioning

confidence: 99%

“…[The Working Group noted that several potential causes of liver cancer, including viral infections (the rates of which are known to be high in the area), 182 IARC MONOGRAPHS VOLUME 92 alcohol consumption and other occupational exposures may have contributed to the excess mortality from liver cancer.] 2.1.8Calcium carbide production Kjuus et al (1986) reported on the incidence of cancer in a cohort of 790 male Norwegian calcium carbide production workers. All workers who had been employed for at least 18 months between 1953 and 1970 were followed for cancer incidence and mortality until 1983.…”

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confidence: 99%

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Integrating Field Analyses with Laboratory Exposures to Assess Ecosystems Health

Hellou¹,

Beach²,

Leonard³

et al. 2012

Polycyclic Aromatic Compounds

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initiated a programme on the evaluation of the carcinogenic risk of chemicals to humans involving the production of critically evaluated monographs on individual chemicals. The programme was subsequently expanded to include evaluations of carcinogenic risks associated with exposures to complex mixtures, life-style factors and biological and physical agents, as well as those in specific occupations. The objective of the programme is to elaborate and publish in the form of monographs critical reviews of data on carcinogenicity for agents to which humans are known to be exposed and on specific exposure situations; to evaluate these data in terms of human risk with the help of international working groups of experts in chemical carcinogenesis Publications of the World Health Organization enjoy copyright protection in accordance with the provisions of Protocol 2 of the Universal Copyright Convention. All rights reserved.The International Agency for Research on Cancer welcomes requests for permission to reproduce or translate its publications, in part or in full. Requests for permission to reproduce or translate IARC publications -whether for sale or for noncommercial distribution -should be addressed to WHO Press, at the above address (fax: +41 22 791 4806; email: permissions@who.int).The designations employed and the presentation of the material in this publication do not imply the expression of any opinion whatsoever on the part of the Secretariat of the World Health Organization concerning the legal status of any country, territory, city, or area or of its authorities, or concerning the delimitation of its frontiers or boundaries.The mention of specific companies or of certain manufacturers' products does not imply that they are endorsed or recommended by the World Health Organization in preference to others of a similar nature that are not mentioned. Errors and omissions excepted, the names of proprietary products are distinguished by initial capital letters.The IARC Monographs Working Group alone is responsible for the views expressed in this publication. IARC Library Cataloguing in Publication Data NOTE TO THE READERThe term 'carcinogenic risk' in the IARC Monographs series is taken to mean that an agent is capable of causing cancer under some circumstances. The Monographs evaluate cancer hazards, despite the historical presence of the word 'risks' in the title.Inclusion of an agent in the Monographs does not imply that it is a carcinogen, only that the published data have been examined. Equally, the fact that an agent has not yet been evaluated in a Monograph does not mean that it is not carcinogenic.The evaluations of carcinogenic risk are made by international working groups of independent scientists and are qualitative in nature. No recommendation is given for regulation or legislation.Anyone who is aware of published data that may alter the evaluation of the carcinogenic risk of an agent to humans is encouraged to make this information available to the Section of IARC Monographs, International Agency for...

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Incidence of cancer among workers producing calcium carbide.

Cited by 20 publications

References 9 publications

Association between diesel exposure at work and prostate cancer

Association between diesel exposure at work and prostate cancer

Calcium Carbide-Induced Haematological Alterations In The Albino Mice- Mus musculus

Integrating Field Analyses with Laboratory Exposures to Assess Ecosystems Health

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