Incidence of patent ductus arteriosus and patent foramen ovale in normal infants

Connuck, David; Sun, Jing Ping; Super, Dennis M.; Kirchner, H. Lester; Fradley, Linda Goetz; Harcar-Sevcik, Rose A.; Salvator, Ann; Singer, Lynn T.; Mehta, Sudhir

doi:10.1016/s0002-9149(01)02214-7

Cited by 51 publications

(37 citation statements)

References 6 publications

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“…Birth Prevalence of CHD Subtypes functional abnormality, whereas it is an abnormality in term infants (21). Furthermore, CHD prevalence highly depends on the sensitivity and specificity of the detection method.…”

Section: Figurementioning

confidence: 99%

Birth Prevalence of Congenital Heart Disease Worldwide

Linde

Konings

Slager

et al. 2011

Journal of the American College of Cardiology

2,740

1,723

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Congenital heart disease (CHD) accounts for nearly one-third of all major congenital anomalies. CHD birth prevalence worldwide and over time is suggested to vary; however, a complete overview is missing. This systematic review included 114 papers, comprising a total study population of 24,091,867 live births with CHD identified in 164,396 individuals. Birth prevalence of total CHD and the 8 most common subtypes were pooled in 5-year time periods since 1930 and in continent and income groups since 1970 using the inverse variance method. Reported total CHD birth prevalence increased substantially over time, from 0.6 per 1,000 live births (95% confidence interval [CI]: 0.4 to 0.8) in 1930 to 1934 to 9.1 per 1,000 live births (95% CI: 9.0 to 9.2) after 1995. Over the last 15 years, stabilization occurred, corresponding to 1.35 million newborns with CHD every year. Significant geographical differences were found. Asia reported the highest CHD birth prevalence, with 9.3 per 1,000 live births (95% CI: 8.9 to 9.7), with relatively more pulmonary outflow obstructions and fewer left ventricular outflow tract obstructions. Reported total CHD birth prevalence in Europe was significantly higher than in North America (8.2 per 1,000 live births [95% CI: 8.1 to 8.3] vs. 6.9 per 1,000 live births [95% CI: 6.7 to 7.1]; p < 0.001). Access to health care is still limited in many parts of the world, as are diagnostic facilities, probably accounting for differences in reported birth prevalence between high- and low-income countries. Observed differences may also be of genetic, environmental, socioeconomical, or ethnic origin, and there needs to be further investigation to tailor the management of this global health problem.

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Section: Figurementioning

confidence: 99%

Birth Prevalence of Congenital Heart Disease Worldwide

Linde

Konings

Slager

et al. 2011

Journal of the American College of Cardiology

2,740

1,723

View full text Add to dashboard Cite

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“…For ASD the situation is just as complex. A study of 102 healthy newborns showed that 62% had an ASD during the first days of life and at follow-up after median 2.3 months, 48 of 89 (55%) still had a left-to-right atrial shunt through an ASD of more than 3 mm [12]. In 113 first time echocardiographies in infants of less than three months, 61 were found to have an inter-atrial communication (secundum ASD) [13].…”

Section: Asdmentioning

confidence: 99%

Atrial and ventricular septal defects – epidemiology and spontaneous closure

Garne

2006

The Journal of Maternal-Fetal & Neonatal Medicine

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“…5,18 It has been suggested that 45% of all infants have a PDA during the first 60 hours of life. 19 A functional PDA is recognized by the presence of a murmur, bounding peripheral pulses, cardiomegaly, increased pulmo- nary fluid, and a hyperactive pericardium, 20 and MPH has strongly been associated with a symptomatic PDA. 21 However, none of the infants we reviewed had any clinical suggestion of a functional PDA.…”

Section: Discussionmentioning

confidence: 99%

Patent Ductus Arteriosus as a Natural Cause of Pulmonary Hemorrhage in Infants

Lewis¹,

McKeever²,

Rutty³

2004

American Journal of Forensic Medicine &Amp; Pathology

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Patent ductus arteriosus (PDA) is a recognized risk factor for massive pulmonary hemorrhage (MPH) in the newborn and is generally seen in association with other MPH risk factors such as prematurity. We report 6 cases of sudden and unexpected death of infants older than 4 days with MPH and PDA at autopsy. The cases were reviewed for other factors that could contribute to MPH to ascertain whether PDA is directly linked to MPH. Histology samples were examined for distribution of hemorrhage in the lungs and iron stained for hemosiderin evaluation. All of the cases had clinical histories and scene examinations which raised the differential diagnosis of mechanical asphyxia in the form of so-called overlayings. The diagnostic dilemma of attributing the MPH to the PDA as the sole cause, dual cause, or incidental finding is discussed. These cases illustrate the medicolegal dilemma faced by the pathologist, as well as the need for further research into the potential association of PDA with MPH.

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Incidence of patent ductus arteriosus and patent foramen ovale in normal infants

Cited by 51 publications

References 6 publications

Birth Prevalence of Congenital Heart Disease Worldwide

Birth Prevalence of Congenital Heart Disease Worldwide

Atrial and ventricular septal defects – epidemiology and spontaneous closure

Patent Ductus Arteriosus as a Natural Cause of Pulmonary Hemorrhage in Infants

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