Indoor passive smoking: its effect on cardiac performance

Leone, Aurelio; Mori, L; Bertanelli, F; Fabiano, Paolo; Filippelli, Mario

doi:10.1016/0167-5273(91)90354-r

Cited by 47 publications

(56 citation statements)

References 4 publications

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“…The determination of exposure to carbon monoxide may be also conducted by measuring the gas in expired air. This determination may be useful, particularly in individuals undergone acute passive smoking exposure [1].…”

Section: Carbon Monoxidementioning

confidence: 99%

Endothelial Dysfunction in Passive Smokers

Leone¹

2014

JCCR

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Section: Carbon Monoxidementioning

confidence: 99%

Endothelial Dysfunction in Passive Smokers

Leone¹

2014

JCCR

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“…In addition, increased systolic blood pressure is an easy parameter to be assessed during acute exposure since it occurs either while an individual smokes a cigarette or during passive acute exposure. Hypoxia due to the increased concentrations of carboxyhemoglobin is responsible for heart alterations consisting of impaired exercise performance in both healthy individuals and individuals suffering from ischemic heart disease [22][23][24][25] together with increased heart rate.…”

Section: Controversies On the Topicmentioning

confidence: 99%

Current and Controversial Features of Cardiovascular Damage from Cigarette Smoking

Leone

2016

Journal of Cardiology and Therapy

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A great number of studies have undoubtedly demonstrated that cardiovascular damage from cigarette smoking cannot be denied, although some controversies on the topic still exist. The damage to the heart and blood vessels is initially characterized by transient functional alterations consisting of endothelial dysfunction, reduced exercise tolerance, increased systolic blood pressure and heart rate following acute exposure. On the contrary, chronic exposure to smoking develops, at the time, firstly reversible and, then, irreversible lesions mainly involving myocardium with an ischemic damage and smoke cardiomyopathy as well as coronary, carotid and cerebral arteries, which display atherosclerotic pathology. The analysis of the epidemiological studies on cardiovascular damage support the controversies on the effects of smoking, because not the same alterations are sometimes observed in different studies on the topic. However, when standardized experimental procedures are followed, the same alterations are clearly reproduced showing that cardiovascular damage from cigarette smoking cannot be denied.

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“…Generally, carbon monoxide from a single cigarette smoked achieves a small concentration in the blood, which determines initially functional but transient effects. However, there is evidence of stronger ischemic responses caused by this chemical related to the hypoxia as documented by the reduced exercise tolerance during smoking exposure [6]. On the contrary, chronic and prolonged exposure shows irreversible alterations of the heart and blood vessels as ischemic heart disease, stroke rate, and coronary and systemic artery lesions consisting of multiple narrowing and thickening of the vascular wall with increased arterial stiffness [7].…”

Section: Editorialmentioning

confidence: 99%

Cardiovascular Risk from Smoking: Doubt or Absolute Certainty?

Leone¹

2015

JCCR

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Indoor passive smoking: its effect on cardiac performance

Cited by 47 publications

References 4 publications

Endothelial Dysfunction in Passive Smokers

Endothelial Dysfunction in Passive Smokers

Current and Controversial Features of Cardiovascular Damage from Cigarette Smoking

Cardiovascular Risk from Smoking: Doubt or Absolute Certainty?

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