1946
DOI: 10.1172/jci101688
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Infectious Hepatitis Complicated by Secondary Invasion With Salmonella 1

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Cited by 19 publications
(3 citation statements)
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“…however, neither the appearance, maintenance, (31,97,128,163,208) Neonatal stage (0-30 days) (12,20,57,175,191) Parenteral iron (8, 65) Increased release of iron from stores Hepatitis (59,85,169) Refeeding starvees (141, 143, (58) cytes Malaria (12,20,126,184) Sickling (7,12,20,27,176) (12,20,57,175,191) metabolism, surface properties, nor permeability functions of the macrophages were altered by the metal. The authors concluded that iron simply was required as a bacterial nutrilite and that "seclusion within tissue cells does not isolate microbes from external nutrients" (206).…”
Section: Effiect Of Iron On Phagocytosis and Immune Responses Iron Exmentioning
confidence: 99%
“…however, neither the appearance, maintenance, (31,97,128,163,208) Neonatal stage (0-30 days) (12,20,57,175,191) Parenteral iron (8, 65) Increased release of iron from stores Hepatitis (59,85,169) Refeeding starvees (141, 143, (58) cytes Malaria (12,20,126,184) Sickling (7,12,20,27,176) (12,20,57,175,191) metabolism, surface properties, nor permeability functions of the macrophages were altered by the metal. The authors concluded that iron simply was required as a bacterial nutrilite and that "seclusion within tissue cells does not isolate microbes from external nutrients" (206).…”
Section: Effiect Of Iron On Phagocytosis and Immune Responses Iron Exmentioning
confidence: 99%
“…Two of the patients included in this series had bacteremia with Sal. cholera suis superimposed on their experimentally induced infectious hepatitis (18). The changes in serum proteins in these two patients were qualitatively similar to the rest of the group, although one of these men had more extreme deflections associated with his more severe illness.…”
Section: Methodsmentioning
confidence: 62%
“…After a spirochaete was discovered as the cause of Weil's disease, extensive investigations were launched to determine if leptospires [47] could be the agent of epidemic catarrhal jaundice: they could not [39]. The typhoid and paratyphoid bacilli were also implicated ; any association between infection by these or other enteric bacteria and acute jaundice was later ascribed to intestinal infections and hepatitis co-prevailing under unsanitary conditions [48].…”
Section: Nosological and Aetiological Clarificationsmentioning
confidence: 99%