Inferior vena cava agenesis in patients with lower limb deep vein thrombosis in the RIETE registry. When and why to suspect

Tufano, Antonella; López-Jiménez, Luciano; Bikdeli, Behnood; García‐Bragado, F; Mazzolai, Lucia; Amitrano, María; Gómez-Cuervo, Covadonga; Marchena, Pablo Javier; Madridano, Olga; Monréal, Manuel; Micco, Pierpaolo Di

doi:10.1016/j.ijcard.2020.01.013

Cited by 21 publications

(25 citation statements)

References 24 publications

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“…The final diagnosis of IVCA is made with subsequent ultrasound, CT or MRI imaging [3] . Other well-reported consequences include skin induration, collateral vein circulation, and most strikingly, non-healing ulcers [4] . Clinical management of IVCA is mostly supportive and preventive with lifelong anticoagulation, minimization of thrombotic risk factors, compression stockings and wound management.…”

Section: Discussionmentioning

confidence: 99%

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Congenital Inferior Vena Cava Agenesis with Ulceration and Deep Vein Thrombosis

Menning

Yousef

2021

European Journal of Case Reports in Internal Medicine

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Objectives: This is one of the few case reports of congenital inferior vena cava atresia (IVCA) with extensive bilateral lower extremity ulcers. Background: IVCA is a congenital anomaly characterized by poor venous return in the lower extremities with increased risk of deep vein thrombi (DVTs) and, rarely, non-healing ulcers. Results: A 45-year-old woman with history significant for DVTs presented with extensive bilateral lower extremity ulcers, and a CT scan revealed congenital IVCA. Oedema management, DVT prevention, routine wound care and skin grafts resolved the ulcers. Conclusion: Congenital venous abnormalities should be included in the differential diagnosis of multiple unprovoked DVTs and non-healing ulcers.

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Section: Discussionmentioning

confidence: 99%

“…Due to the development of collaterals, these malformations can remain asymptomatic for a long period of time. If detected late or managed insufficiently, the impaired blood flow confers a 10-fold increase in the risk of a DVT which can later present with extensive, non-healing ulceration [3,4] .…”

Section: Introductionmentioning

confidence: 99%

Congenital Inferior Vena Cava Agenesis with Ulceration and Deep Vein Thrombosis

Menning

Yousef

2021

European Journal of Case Reports in Internal Medicine

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“…Inferior vena cava anomalies have been recognized as risk factors for deep venous thrombosis (DVT) in children and young adults [8][9][10]. IVC anomalies occur in about 0.5% to 1% of the general population, and in 2% to 3% of patients with congenital heart defects [4,9,10].…”

Section: Etiology Anatomy and Pathophysiologymentioning

confidence: 99%

“…Chee YL et al and Ruggeri M et al reported a prevalence of about 5% in younger DVT-patients [9,10] and Bass JE et al identified 5 (16%) anomalous IVC in 31 younger patients with ilio-femoral DVT assessed by venography and magnetic resonance angiography [5]. Furthermore, analysis of the RIETE-registry revealed that patients with IVC-abnormalities are more likely to develop severe post-thrombotic syndrome after their first DVT than patients without IVC-anomalies [8].Thrombosis of the IVC was otherwise identified to be associated with primary thrombophilia, hormonal contraception, estrogen replacement therapy, Behcet's disease, paroxysmal nocturnal hemoglobinuria, and malignancies [11,12]. IVC-filters increase the risk of IVC-thrombosis especially when anticoagulation therapy is withheld [11][12][13].…”

Section: Etiology Anatomy and Pathophysiologymentioning

confidence: 99%

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Inferior vena cava-syndrome

Klein-Weigel

Elitok

Ruttloff

et al. 2021

Vasa

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Summary: Inferior vena cava syndrome (IVCS) is caused by agenesis, compression, invasion, or thrombosis of the IVC, or may be associated with Budd-Chiari syndrome. Its incidence and prevalence are unknown. Benign IVCS is separated from malignant IVCS. Both cover a wide clinical spectrum reaching from asymptomatic to highly symptomatic cases correlated to the underlying cause, the acuity, the extent of the venous obstruction, and the recruitment and development of venous collateral circuits. Imaging is necessary to determine the underlying cause of IVCS and to guide clinical decisions. Interventional therapy has changed the therapeutic approach in symptomatic patients. This article provides an overview over IVCS and focuses on interventional therapeutic methods and results.

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Bilateral proximal deep vein thrombosis and COVID‐19 in a patient with absence of inferior vena cava: A case report and review of literature

Jenab

Ghafouri

Hosseini

et al. 2022

Clinical Case Reports

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Bilateral proximal deep vein thrombosis (DVT) in the lower extremities of young patients should raise suspicion over pro-thrombotic conditions and venous anatomical abnormalities, even in the presence of other precipitating factors, such as viral infection. The authors present a 33-year-old man with bilateral DVT and absence of inferior vena cava (AIVC), who also had concurrent COVID-19, and discuss the management of this patient. K E Y W O R D Sabsence of inferior vena cava, anticoagulants, deep vein thrombosis, inferior vena cava agenesis, inferior vena cava atresia

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Inferior vena cava agenesis in patients with lower limb deep vein thrombosis in the RIETE registry. When and why to suspect

Cited by 21 publications

References 24 publications

Congenital Inferior Vena Cava Agenesis with Ulceration and Deep Vein Thrombosis

Congenital Inferior Vena Cava Agenesis with Ulceration and Deep Vein Thrombosis

Inferior vena cava-syndrome

Bilateral proximal deep vein thrombosis and COVID‐19 in a patient with absence of inferior vena cava: A case report and review of literature

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