“…For example, within the nasal region, neural cell adhesion molecules (NCAM; Gong and Shipley, 1996), netrins (Shu et al, 2000), semaphorins (Gavazzi, 2001), EphB2 (St. John and Key, 2001), NELF (Kramer and Wray, 2000a), and CCK (Giacobini et al, 2004) have been identified to influence, either directly or indirectly, olfactoryvomeronasal axon outgrowth and GnRH-1 neuronal migration. At the nasal forebrain junction/cribriform plate, new molecular players appear, i.e., GABA (Fueshko et al, 1998;Bless et al, 2000), glutamate (Simonian and Herbison, 2001), and serotonin (Pronina et al, 2003); while others disappear, i.e., NELF (Kramer and Wray, 2000a). Several of these molecules have been proposed to regulate entrance of the GnRH-1 neurons into the developing forebrain (Wray, 2002).…”