“…According to data from the literature, the aquaporins of prokaryotes, archaea and eukaryotes differ in the specificity of pores to transferred substrates, according to [19][20][21][22][23]. The secondary stage of the response to salt stress occurs as a result of the accumulation of organic substances, such as trehalose, proline, ectoine, glycine betaine or choline (osmolytes [4,[24][25][26], osmoprotectants [27][28][29] or osmoprotectors [26,[30][31][32]), in the cytoplasm of bacteria, which is necessary to maintain cell turgor (prolonged exposure to hyperosmotic conditions) [33]. It should be noted that the data discussed above were obtained exclusively for model strains of bacteria from different species, classes or even orders, while for root-nodule bacteria, symbionts of leguminous plants, data about genes involved in the response to salt stress as well as about their functional role are extremely limited.…”