Intellectual Disability

Shapiro, Bruce K.; Batshaw, Mark L.

doi:10.1016/b978-1-4377-0755-7.00033-6

Cited by 16 publications

(18 citation statements)

References 13 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Standard definitions were used to define cerebral palsy, epilepsy, intellectual disability. (9,10,11) Visual and hearing impairment were assessed by trained ophthalmologist and audiologist and confirmed by performing appropriate tests. Behavioural problems were assessed by the clinical psychologist.…”

Section: Sampling and Data Collectionmentioning

confidence: 99%

Assessment of Quality of Life of Parents of Children With Cerebral Palsy

Chalipat¹,

Malwade²,

Karambelkar³

et al. 2016

jebmh

View full text Add to dashboard Cite

BACKGROUNDRaising a child with chronic disability is stressful and demanding task which can affect the quality of life of the parent/caregiver adversely. The aim of this study was to assess the quality of life of parents of children aged between 2-12 years with cerebral palsy. MATERIALS AND METHODSThis was a cross sectional descriptive questionnaire based study, which included 30 parents of children with cerebral palsy between 2-12 years of age attending Paediatric OPD or Rehabilitation services. Interviews were conducted using a semistructured questionnaire and quality of life was assessed using World Health Organization Quality of Life-BREF (WHOQOL-BREF-) Questionnaire-short version. Statistical analysis was done with descriptive statistics using SPSS version 10. RESULTSThe mean score of the social domain of quality of life of caregivers was the lowest (9.33±1.49 SD) followed by psychological (18.23±1.50 SD). Seventy percent of parents reported their overall quality of life as neither good nor poor and 66.67% of parents were satisfied with their own health. CONCLUSIONQuality of life of parents of children with CP was affected in all domains, with predominant affection of social domain. More than half of parents were satisfied with their own general health and they reported overall quality of life was neither good nor poor. HOW TO CITE THIS ARTICLE:Chalipat S, Malwade SD, Karambelkar GR, et al. Assessment of quality of life of parents of children with cerebral palsy.

show abstract

Section: Sampling and Data Collectionmentioning

confidence: 99%

Assessment of Quality of Life of Parents of Children With Cerebral Palsy

Chalipat¹,

Malwade²,

Karambelkar³

et al. 2016

jebmh

View full text Add to dashboard Cite

show abstract

Section: Introductionmentioning

confidence: 99%

“…Although there are many risk factors, a specific cause is identified for less than half of those with mild disabilities (IQ range 50–69) who make up the majority of cases [ 3 , 10 ]. Mild intellectual disability often clusters within families [ 10 ] suggesting that genetic or other shared familial factors may influence risk. When disabilities are more severe, specific causes are identified in over 75% of cases, often involving genetic or chromosomal abnormalities and inborn errors of metabolism [ 10 ].…”

Section: Introductionmentioning

confidence: 99%

“…Mild intellectual disability often clusters within families [ 10 ] suggesting that genetic or other shared familial factors may influence risk. When disabilities are more severe, specific causes are identified in over 75% of cases, often involving genetic or chromosomal abnormalities and inborn errors of metabolism [ 10 ]. When intellectual disability is present without a specific genetic or chromosomal cause, it is associated with advanced maternal age, maternal risk behaviors or medical problems during pregnancy and fetal growth restriction [ 11 ], suggesting that these may be risk factors.…”

Section: Introductionmentioning

confidence: 99%

See 1 more Smart Citation

Gestational age at birth and risk of intellectual disability without a common genetic cause

et al. 2017

View full text Add to dashboard Cite

Preterm birth is linked to intellectual disability and there is evidence to suggest post-term birth may also incur risk. However, these associations have not yet been investigated in the absence of common genetic causes of intellectual disability, where risk associated with late delivery may be preventable. We therefore aimed to examine risk of intellectual disability without a common genetic cause across the entire range of gestation, using a matched-sibling design to account for unmeasured confounding by shared familial factors. We conducted a population-based retrospective study using data from the Stockholm Youth Cohort (n = 499,621) and examined associations in a nested cohort of matched outcome-discordant siblings (n = 8034). Risk of intellectual disability was greatest among those born extremely early (adjusted OR24 weeks = 14.54 [95% CI 11.46–18.44]), lessening with advancing gestational age toward term (aOR32 weeks = 3.59 [3.22–4.01]; aOR37 weeks = 1.50 [1.38–1.63]); aOR38 weeks = 1.26 [1.16–1.37]; aOR39 weeks = 1.10 [1.04–1.17]) and increasing with advancing gestational age post-term (aOR42 weeks = 1.16 [1.08–1.25]; aOR43 weeks = 1.41 [1.21–1.64]; aOR44 weeks = 1.71 [1.34–2.18]; aOR45 weeks = 2.07 [1.47–2.92]). Associations persisted in a cohort of matched siblings suggesting they were robust against confounding by shared familial traits. Risk of intellectual disability was greatest among children showing evidence of fetal growth restriction, especially when birth occurred before or after term. Birth at non-optimal gestational duration may be linked causally with greater risk of intellectual disability. The mechanisms underlying these associations need to be elucidated as they are relevant to clinical practice concerning elective delivery around term and mitigation of risk in post-term children.Electronic supplementary materialThe online version of this article (10.1007/s10654-017-0340-1) contains supplementary material, which is available to authorized users.

show abstract

“…Developmental delay was defined as development quotient (DQ) ≤70 (≤2SD) in either the mental or motor scale. Samples from both groups were further classified as mild, moderate, and severe delay [3]. All the children were further assessed in all the clusters of both domains to evaluate for the specific areas of development affected by malnutrition [4,5].…”

Section: Methodsmentioning

confidence: 99%

Neurodevelopmental Status of Children aged 6-30 months with Severe Acute Malnutrition

Dwivedi

Singh

et al. 2018

Indian Pediatr

View full text Add to dashboard Cite

Objective This cross sectional study was done to assess the developmental status in children (6-30 mo old) with severe acute malutrition (SAM). Methods Study subjects were enrolled from children in SAM therapeutic unit, and controls were selected from well-baby clinic of the institute. Neurodevelopment of both groups was assessed using the Developmental assessment scale of Indian infants (DASII). Developmental quotient (DQ) ≤70 was considered delayed. Results Mean (SE) motor DQ 59.04 (0.74) and mental DQ 62.1 (0.57) was lower in SAM as compared to controls (both P<0.0001). Clusters of early age were normal but clusters with items of later infancy were delayed. Conclusions Children with SAM show significant delay in development, and motor DQ is affected more than mental DQ.

show abstract

Intellectual Disability

Cited by 16 publications

References 13 publications

Assessment of Quality of Life of Parents of Children With Cerebral Palsy

Assessment of Quality of Life of Parents of Children With Cerebral Palsy

Gestational age at birth and risk of intellectual disability without a common genetic cause

Neurodevelopmental Status of Children aged 6-30 months with Severe Acute Malnutrition

Contact Info

Product

Resources

About