Abstract:Animal lipoxygenases (EC 1.13.11.12, LOX) convert arachidonic acid to biological active compounds known as eicosanoids. Proinflammatory
eicosanoids - leukotrienes are involved in progression of atherosclerosis. Our attention was focused on a connection
between activity of LOX and primary hypertriacylglycerolaemia, untreated or treated with fenofibrate. Our results show that
LOX activity in the cytoplasm derived from liver of hypertriacylglycerolaemic rats is significantly increased (pH 7,2; relative to the
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