Long-term histological evaluation of the natural history and prognostic factors of alcoholic liver disease

Marbet, Urs A.; Bianchi, Leonardo; Meury, U.; Stalder, G

doi:10.1016/s0168-8278(87)80547-0

Cited by 109 publications

(58 citation statements)

References 30 publications

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“…Disagreement exists, however, regarding whether the amount of alcohol ingested increases the risk of alcohol liver disease proportionally (69,70).…”

Section: Risk Factorsmentioning

confidence: 99%

Alcoholic liver disease: proposed recommendations for the American College of Gastroenterology

McCullough

1998

The American Journal of Gastroenterology

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PREAMBLEGuidelines for clinical practice are intended to suggest preferable approaches to particular medical problems as established by interpretation and collation of scientifically valid research, derived from extensive review of published literature. When data are not available that will withstand objective scrutiny, a recommendation may be made based on a consensus of experts. Guidelines are intended to apply to the clinical situation for all physicians without regard to specialty. Guidelines are intended to be flexible, not necessarily indicating the only acceptable approach, and should be distinguished from standards of care that are inflexible and rarely violated. Given the wide range of choices in any health care problem, the physician should select the course best suited to the individual patient and the clinical situation presented.These guidelines are developed under the auspices of the American College of Gastroenterology and its Practice Parameters Committee. These guidelines are also approved by the governing boards of the American Gastroenterological Association and the American Association for the Study of Liver Disease. Expert opinion is solicited from the outset for the document. Guidelines are reviewed in depth by the Committee, with participation from experienced clinicians and others in related fields. The final recommendations are based on the data available at the time of the production of the document and may be updated with pertinent scientific developments at a later time.Objective: The objective of this work was to develop practice guidelines for the management of alcoholic liver disease. Method: A computerized search using the Medline Data Base from 1966 -July 1997 was performed with the search headings; alcohol, alcoholic hepatitis, alcoholic liver disease, liver transplant, diagnosis, epidemiology, human, and English only. All randomized controlled trials, case-control studies, and meta-analyses were read in depth. A manual search was also done using references from each retrieved report, review articles, editorials, postgraduate course syllabi, and textbooks. In the subsequent review, evidence was evaluated using a hierarchical scale with randomized, controlled trials given the most importance. Abstracts presented at national meetings were included only when unique data were obtained from those studies. (Am J Gastroenterol 1998;93:2022-2036.

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“…Disagreement exists, however, regarding whether the amount of alcohol ingested increases the risk of alcohol liver disease proportionally (69,70).…”

Section: Risk Factorsmentioning

confidence: 99%

Alcoholic liver disease: proposed recommendations for the American College of Gastroenterology

McCullough

1998

The American Journal of Gastroenterology

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“…in all of 6 alcoholic patients with CAH despite lowered alcohol consumption, suggesting that immunological factors were responsible for the progre~sion.~~ In their follow-up study, however, the histological features of a classic alcoholic hepatitis were initially observed in 2 of the 6 patients whose subsequent histologies were indeed CAH, while the other 4 patients showed no evidence of alcoholic hepatitis during the observation period. 45 Thus, in addition to alcoholic liver fibrosis, the chronic hepatitis seen in alcoholic patien1.s and aetiologically defined as alcoholic may be involved in the development of alcoholic cirrhosis, as has been widely recognized in European and North American countries in patients with alcoholic hepatitis. In view of the aetiological role of alcoholic cirrhosis, however, the extent of the influence of liver fibrosis and chronic hepatitis remains to be determined.…”

Section: Possible Precursor Lesions Of Alcoholic Cirrhosismentioning

confidence: 99%

Alcoholic liver disease in Japanese patients: A comparison with Caucasians

Hasumura

Takeuchi

1991

J of Gastro and Hepatol

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“…3 Other studies of patients with alcoholic fatty liver diseases are small (fewer than 100 patients) but suggest that women have a higher cirrhosis risk than men. [4][5][6] Moreover, the mortality of patients with alcoholic fatty liver disease is markedly higher than in the general population, 7,8 but their actual risk of death within a specified time interval is yet unreported. Alcoholic fatty liver disease comprises alcoholic pure steatosis characterised by macrovesicular steatosis and alcoholic steatohepatitis characterised by neutrophil infiltration, ballooning degeneration, Mallory bodies and variable fibrosis.…”

Section: Introductionmentioning

confidence: 99%

Cirrhosis and mortality risks of biopsy‐verified alcoholic pure steatosis and steatohepatitis: a nationwide registry‐based study

Deleuran

Grønbæk

Vilstrup

et al. 2012

Aliment Pharmacol Ther

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Long-term histological evaluation of the natural history and prognostic factors of alcoholic liver disease

Cited by 109 publications

References 30 publications

Alcoholic liver disease: proposed recommendations for the American College of Gastroenterology

Alcoholic liver disease: proposed recommendations for the American College of Gastroenterology

Alcoholic liver disease in Japanese patients: A comparison with Caucasians

Cirrhosis and mortality risks of biopsy‐verified alcoholic pure steatosis and steatohepatitis: a nationwide registry‐based study

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