Magnetic resonance imaging of intracranial venous angiomas

Uchino, Akira; Imada, Hajime; Ohno, Masato

doi:10.1016/0899-7071(90)90044-c

Cited by 13 publications

(10 citation statements)

References 8 publications

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“…The penitoneal cavity contained ascites fluid and thick layers of putrid and [4]; in that case, they were associated with adenopathy in the porta hepatis, which we did not find in our patient. Also, the size and location of the pulmonary lesions in our patient were not typical for lesions associated with either miliary or granulomatous seeding.…”

Section: Abdominal Tuberculosis Mimicking a Gynecologic Tumorcontrasting

confidence: 42%

“…He used the word diverticulum, which may have led to the confusion. The word ampulla does not appear in his article [4]. Q is the symbol for blood volume (not flow) and may well have come from Quellung.…”

Section: Lettersmentioning

confidence: 99%

“…Unfortunately, these neoplasms are often detected and diagnosed at a late stage, when they are incurable [1]. Extensive spread ofthe tumor frequently involves adjacent vital organs [4]. Hematogenous metastases to the liven and lungs are common [1 , 3].…”

Section: Leiomyosarcoma Of the Pancreatic Bed Detected On Ct Scansmentioning

confidence: 99%

“…3]. Complete surgical excision is the primary treatment of netropenitoneal sarcoma and offers the best chance for cure [4]. Unfortunately, these neoplasms are often detected and diagnosed at a late stage, when they are incurable [1].…”

Section: Leiomyosarcoma Of the Pancreatic Bed Detected On Ct Scansmentioning

confidence: 99%

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Daves¹,

Zeligman²

1993

American Journal of Roentgenology

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Section: Abdominal Tuberculosis Mimicking a Gynecologic Tumorcontrasting

confidence: 42%

Section: Lettersmentioning

confidence: 99%

Section: Leiomyosarcoma Of the Pancreatic Bed Detected On Ct Scansmentioning

confidence: 99%

Section: Leiomyosarcoma Of the Pancreatic Bed Detected On Ct Scansmentioning

confidence: 99%

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Daves¹,

Zeligman²

1993

American Journal of Roentgenology

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“…We analyzed epidemio- We restricted this review to complications considered to be directly related to DVAs to recognize under which conditions they could become symptomatic. The following exclusion criteria were therefore chosen: (1) unspecific symptoms like headaches or longstanding symptoms such as epilepsy were not considered if there was no MR evidence of a causative link to the DVA (such as congestive edema in the immediate vicinity); 7,9 -14 (2) MRI abnormalities without symptoms (T2* hypointensities, T2/flair hyperintensity), although related to DVAs, were not included; 5,15,16 (3) patients with DVAs and symptomatic cavernomas because of their established natural history (hemorrhage, epilepsy, mass effect); 3,14,17-24 and (4) patients and series 25,26 presenting incomplete radiological or clinical data necessary to exclude completely an associated disease (eg, patients investigated or manuscripts before the MRI era).…”

Section: Methodsmentioning

confidence: 99%

Pathomechanisms of Symptomatic Developmental Venous Anomalies

Pereira

Geibprasert

Krings

et al. 2008

Stroke

180

182

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Background and Purpose-Although it is generally accepted that developmental venous anomalies (DVAs) are benign vascular malformations, over the past years, we have seen patients with symptomatic DVAs. Therefore, we performed a retrospective study and a literature study to review how, when, and why DVAs can become clinically significant. Methods-Charts and angiographic films of 17 patients with DVAs whose 18 vascular symptoms could be attributed to a DVA were selected from a neurovascular databank of our hospital. MRI had to be available to rule out any other associated disease. In the literature, 51 cases of well-documented symptomatic DVAs were found. Pathomechanisms were divided into mechanical and flow-related causes. Results-Mechanical (obstructive or compressive) pathomechanisms accounted for 14 of 69 symptomatic patients resulting in hydrocephalus or nerve compression syndromes. Flow-related pathomechanisms (49 of 69 patients) could be subdivided into complications resulting from an increase of flow into the DVA (owing to an arteriovenous shunt using the DVA as the drainage route; nϭ19) or a decrease of outflow (nϭ26) or a remote shunt with increased venous pressure (nϭ4) leading to symptoms of venous congestion. In 6 cases, no specific pathomechanisms were detected. Conclusions-Although DVAs should be considered benign, under rare circumstances, they can be symptomatic. DVAs, as extreme variations of normal venous drainage, may represent a more fragile venous drainage system that can be more easily affected by in-and outflow alterations. The integrity of the DVA needs to be preserved irrespective of the treatment that should be tailored to the specific pathomechanism. (Stroke. 2008;39:3201-3215.)

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Slow-Flow, Asymptomatic Vascular Malformations: Brain Capillary Telangiectasias and Developmental Venous Anomalies

McKinney

2017

Atlas of Normal Imaging Variations of the Brain, Skull, and Craniocervical Vasculature

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Magnetic resonance imaging of intracranial venous angiomas

Cited by 13 publications

References 8 publications

What's your forte?

What's your forte?

Pathomechanisms of Symptomatic Developmental Venous Anomalies

Slow-Flow, Asymptomatic Vascular Malformations: Brain Capillary Telangiectasias and Developmental Venous Anomalies

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