“…When pressurizing the LV of an arrested heart, the MV apparatus and subvalvular structures dilate in a state similar to that of diastole when the LV would otherwise be contracting during systole in a beating heart. 12 , 13 More specifically, in vivo, the PMs, which commonly serve as the distal anchor point of the neochordae, move with each heartbeat, translating and rotating relative to the valve annulus, and this movement can be generally characterized as a reduction in the distance between the PM and the valve annulus upon leaflet coaptation. 14 , 15 On the contrary, static pressurization simulates the opposite as it causes the LV to dilate, moving the PMs further away from the annulus upon leaflet coaptation.…”