Meconium Peritonitis: A Case Study

Abstract

Meconium peritonitis is a sterile chemical peritonitis preceded by bowel perforation, resulting in meconium leakage and subsequent inflammatory cascade within the peritoneal cavity. The presentation can range from simple failure of the neonate to pass meconium to complications such as persistent pulmonary hypertension, lung hypoplasia, and systemic inflammatory syndrome. The purpose of this article is to review a case of meconium peritonitis while considering its etiology, diagnosis, management, and multidisciplinary team care.

Fetal Abdominal Cyst as a Stage of Meconium Peritonitis after Fetoscopic Laser Photocoagulation

“…The most common ultrasonographic findings include bowel dilatation, intraabdominal calcification, ascites, intraperitoneal pseudocyst, and polyhydramnios. 5,6,10,[14][15][16] Our case is quite unusual because of its unusual course starting from ascites and hyperechoic bowel, presenting further with intraperitoneal pseudocyst with intestinal connection, intraperitoneal calcifications, dilated aperistaltic bowel loops, and polyhydramnion. Pseudocyst was not seen later; a possible explanation is the formation of intestinal fistula.…”

“…The most common ultrasonographic findings include bowel dilatation, intraabdominal calcification, ascites, intraperitoneal pseudocyst, and polyhydramnios. 5 6 10 14 15 16…”

Fetal Meconium Peritonitis: A Clinical Study of Nine Cases

“…In the present study, the most common ultrasonographic findings were fetal intestinal dilatation, followed by fetal intraperitoneal calcification, fetal ascites with intestinal perforation and dilation of the oop intestine, and 1 case presenting with hyperhyniotic amniotic fluid. Some researchers classify FMP into three types depending on the presence of specific features, with type I FMP presenting with ascites, type II FMP being associated with cysts as a consequence of local ascites accumulation and adhesion in the bowel and greater omentum having resulted in pseudocyst formation, and type III FMP being associated with fibrous adhesions as a consequence of calcium salt closed perforation caused by the sedimentation of the bowel perforation position [ 4 , 5 , 12 ]. With intraperitoneal calcification as the common phenomenon, Zangheri et al divided FMP into four levels according to the presence of other abnormal ultrasonographic situations: level 0 refers to the cases where only intraperitoneal calcification is observed, level 1 refers to the cases where intraperitoneal calcification and one abnormal ultrasonographic situation are observed, level 2 refers to the cases where intraperitoneal calcification and two abnormal ultrasonographic situations are observed, and level 3 refers to the cases where intraperitoneal calcification and three abnormal ultrasonographic situations are observed [ 7 ].…”