Melanin depigmentation of the palatal mucosa in reverse smokers: a preliminary study

Hedin, C; Pindborg, J. J.; Daftary, D. K.; Mehta, Fali S.

doi:10.1111/j.1600-0714.1992.tb00971.x

Cited by 18 publications

(18 citation statements)

References 15 publications

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“…4 Smoker's melanosis occurs in up to 21.5% of smokers. 1 Smoking may cause increased production of melanin, which may provide a biologic defence against the noxious agents present in tobacco smoke 5 and it is hypothesized that melanin may play a role in detoxification of polycyclic amines, nicotine and benzopyrene. The intensity of the pigmentation is related to the duration and amount of smoking.…”

Section: Discussionmentioning

confidence: 99%

Oral Melanosis

Kumar¹

2011

International Journal of Head and Neck Surgery

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There is very little information in literature about oral melanosis not associated with racial pigmentation or secondary to other syndromes. Various stimuli that can result in an increased production of melanin at the level of mucosa include trauma, hormones, radiation and medications. Three such cases are reported in which stimulus for genesis of melanosis was mechanical trauma in form of smoking.

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Section: Discussionmentioning

confidence: 99%

Oral Melanosis

Kumar¹

2011

International Journal of Head and Neck Surgery

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“…stated that smoking may cause oral pigmentation in light-skinned individuals and accentuate the pigmentation of dark skinned patients [18]. There is increased production of melanin, which may provide a biologic defense against the noxious agents present in tobacco smoke [19]. Smoker's melanosis occurs in up to 21.5% of smokers [20].…”

Section: Chemically Induced Melanosismentioning

confidence: 99%

Diagnosis of oral pigmentations and malignant transformations

Tarakji

Umair

Prasad

et al. 2014

Singapore Dental Journal

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Oral pigmentation may present as focal, multifocal or diffused macular or tumefactive lesions. They may greatly vary in color as blue, purple, brown, gray or black depending on the quantity and site of melanin in the tissues [1]. Etiology of pigmentation can be multi factorial. Mostly pigmentation is physiologic but at times it can be a precursor of severe diseases. Lesions may be caused by localized harmless accumulations of melanin, hemosiderin or exogenous metals or they may be a sign of underlying systemic or genetic disease. A few lesions may be associated with life-threatening medical conditions that require immediate intervention. The differential diagnosis for any pigmented lesion is extensive, as it includes examples of endogenous and exogenous pigmentations. Although biopsy is a helpful and necessary aid in the diagnosis of focally pigmented lesions, with diffuse pigmentation lesions require a thorough dental and medical history and laboratory investigations.

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“…Smoking was found to cause diffuse oral melanin pigmentation in European and Asian populations 21‐24 . Increased pigmentation was reported in 21.5% of smokers, and the intensity of the pigmentation was related to the number of cigarettes consumed 21 .…”

mentioning

confidence: 99%