Migraine Accompanied by Hemifacial Spasm: A Case Report

Husid, Marc S.

doi:10.1111/j.1526-4610.2005.05053_3.x

Cited by 11 publications

(10 citation statements)

References 12 publications

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“…At least two patients with concurrent HFS and ipsilateral migraine have been observed: that reported by Husid (5) and the one reported here. A pathophysiological link rather than a coincidental co-occurrence seems likely, since both phenomena have been coupled for many years in each patient.…”

Section: Discussionsupporting

confidence: 62%

“…In fact, the appearance of abnormal movements is not included among the potential features of migraine attacks in the International Headache Society classification (4). Recently, the case of a woman having repeated episodes of concurrent hemifacial spasm (HFS) and migraine has been reported (5). We also describe here a patient with unilateral migraine associated with ipsilateral HFS and provide some additional findings that might point to possible pathophysiological mechanisms.…”

mentioning

confidence: 99%

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Hemifacial Spasm Induced by Trigeminal Stimuli and Coupled With Ipsilateral Migraine: A Spasmodic Migraine

Cuadrado

Fernández‐de‐las‐Peñas

Santiago

et al. 2006

Cephalalgia

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Section: Discussionsupporting

confidence: 62%

mentioning

confidence: 99%

Hemifacial Spasm Induced by Trigeminal Stimuli and Coupled With Ipsilateral Migraine: A Spasmodic Migraine

Cuadrado

Fernández‐de‐las‐Peñas

Santiago

et al. 2006

Cephalalgia

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“…Cluster headache is postulated to be associated with an abnormal interaction between trigeminal afferents and cranial parasympathetic efferents. Trigeminal afferent input alters the excitability of facial motoneurons in HFS 67–69. In our series, 6 patients had associated migraine, and 1 patient had trigeminal neuralgia, possibly indicative of increased excitability of brain stem nuclei.…”

Section: Discussionmentioning

confidence: 62%

The many faces of hemifacial spasm: Differential diagnosis of unilateral facial spasms

2011

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Hemifacial spasm is defined as unilateral, involuntary, irregular clonic or tonic movement of muscles innervated by the seventh cranial nerve. Most frequently attributed to vascular loop compression at the root exit zone of the facial nerve, there are many other etiologies of unilateral facial movements that must be considered in the differential diagnosis of hemifacial spasm. The primary purpose of this review is to draw attention to the marked heterogeneity of unilateral facial spasms and to focus on clinical characteristics of mimickers of hemifacial spasm and on atypical presentations of nonvascular cases. In addition to a comprehensive review of the literature on hemifacial spasm, medical records and videos of consecutive patients referred to the Movement Disorders Clinic at Baylor College of Medicine for hemifacial spasm between 2000 and 2010 were reviewed, and videos of illustrative cases were edited. Among 215 patients referred for evaluation of hemifacial spasm, 133 (62%) were classified as primary or idiopathic hemifacial spasm (presumably caused by vascular compression of the ipsilateral facial nerve), and 4 (2%) had hereditary hemifacial spasm. Secondary causes were found in 40 patients (19%) and included Bell's palsy (n=23, 11%), facial nerve injury (n=13, 6%), demyelination (n=2), and brain vascular insults (n=2). There were an additional 38 patients (18%) with hemifacial spasm mimickers classified as psychogenic, tics, dystonia, myoclonus, and hemimasticatory spasm. We concluded that although most cases of hemifacial spasm are idiopathic and probably caused by vascular compression of the facial nerve, other etiologies should be considered in the differential diagnosis, particularly if there are atypical features.

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“…It has been suggested that migraine could map or remap to a site of trigeminal nerve injury (11,12). Thus, in rare instances, the origin of the trigeminal nerve might become involved by the migraine mechanism, as can occur with the oculomotor nerves in ophthalmoplegic migraine (13) and the facial nerve in migrainous facial palsy and hemifacial spasm (14–16). These two patients suggest a reciprocal link, with the migraine mechanism activating the trigeminal nerve to facilitate neuralgic pain and vice versa.…”

Section: Discussionmentioning

confidence: 99%

Migraine-tic syndrome: Two further case reports

et al. 2016

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Background Migraine-tic syndrome was first reported in 2004 in a 44-year-old woman who had concomitant symptoms of both typical trigeminal neuralgia and migraine. We report here two further cases of migraine-tic syndrome and speculate on the relevance of this condition to the pathophysiology of headache. Case reports A 43-year-old woman presented with typical trigeminal neuralgia symptoms that preceded the onset of migraine headache; both headache types responded to treatment with sumatriptan. A 35-year-old woman presented with trigeminal neuralgia that consistently followed the onset of migraine headache. The former aspect responded to baclofen, but the migraine headache required treatment with amitriptyline. Discussion These two patients provide further support for the presence of an overlap syndrome of migraine-tic. We suggest that there is a common pathway for trigeminal neuralgia and migraine.

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Migraine Accompanied by Hemifacial Spasm: A Case Report

Abstract: A 36-year-old woman has had repeated episodes of concurrent hemifacial spasm and migraine for 8 years. Her history is summarized and possible pathophysiologic links are discussed.

Cited by 11 publications

References 12 publications

Hemifacial Spasm Induced by Trigeminal Stimuli and Coupled With Ipsilateral Migraine: A Spasmodic Migraine

Hemifacial Spasm Induced by Trigeminal Stimuli and Coupled With Ipsilateral Migraine: A Spasmodic Migraine

The many faces of hemifacial spasm: Differential diagnosis of unilateral facial spasms

Migraine-tic syndrome: Two further case reports

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