“…The model (2.4) is an extension of numerous malaria transmission models that assess the impact of insecticide resistance and dynamics of resistant vectors when chemical insecticides (such as ITNs, IRS, or larvacides) are used to control the vector population (such as those in (Barbosa & Hastings, 2012; Birget & Koella, 2015; Blayneh & Mohammed-Awel, 2014; Brown et al., 2013; Gourley et al., 2011; Wairimu & Ronoh, 2016)) by, inter alia ,- including the use of ITNs and IRS control strategies where the vector population is stratified according to type (i.e., wild or resistant to insecticides). These classifications are not included in the genetic models in (Barbosa & Hastings, 2012; Birget & Koella, 2015; Brown et al., 2013);
- incorporating mosquito feeding and resting behavior (these are not included in the models in (Barbosa & Hastings, 2012; Birget & Koella, 2015; Blayneh & Mohammed-Awel, 2014; Brown et al., 2013; Gourley et al., 2011; Wairimu & Ronoh, 2016));
- carrying out optimal control analysis of the model with respect to the two insecticide-based controls (this is not done in (Barbosa & Hastings, 2012; Birget & Koella, 2015; Blayneh & Mohammed-Awel, 2014; Brown et al., 2013; Gourley et al., 2011; Wairimu & Ronoh, 2016)).
…”