“…Since no significant (p b 0.05) differences were observed between the efficiencies of recovery of GI and GII, the lack of samples positive for NoV GII could be attributed to a higher environmental stability of the former (da Silva et al, 2007;Pérez-Sautu et al, 2012). This differential survival is considered to be one of the causes of the frequent involvement of GI in outbreaks transmitted by food or water (Arvelo et al, 2012;Blanton et al, 2006;Borchardt et al, 2012;Le Guyader et al, 2012;Matthews et al, 2012;Maunula et al, 2009). As a matter of fact, in one of the few studies reporting the molecular epidemiology of NoV in Egypt, 85% among all strains detected in sewage samples were GI, while in clinical samples GI accounted for 29% of the isolates (Kamel et al, 2010).…”