Occurrence of Cerebral Venous Sinus Thrombosis in Patients with Presumed Idiopathic Intracranial Hypertension

Lin, Albert P.; Foroozan, Rod; Danesh‐Meyer, Helen V.; Salvo, Gabriella De; Savino, Peter J.; Sergott, Robert C.

doi:10.1016/j.ophtha.2006.05.065

Cited by 94 publications

(53 citation statements)

References 20 publications

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“…MR imaging and MR venography are the most sensitive techniques in determining CVST. In this technique, there is no sign of flow in the occluded vein or sinus and the parenchymal changes can also be evaluated (5,8,11,14). We detected hemorrhagic areas at the left parietal lobe in the MRI examination of our case and did not detect any sinovenous and ipsilateral extracranial internal jugular venous blood flow in several intracranial segments in the MR venography examination.…”

Section: Discussionmentioning

confidence: 57%

“…The diagnosis can easily be made by using BT and MR imaging and obtaining a proper clinical history. BT is the first imaging modality and thrombosis is manifested as hyperdensity on non-contrast BT (5,8,11,14). In the first BT of our case, there were dilated hyperdense images at the left cortical vein at the vertex level, and there also were hyperdense hemorrhagic areas, which have peripheral hypodense edematous areas in the left parietal lobe.…”

Section: Discussionmentioning

confidence: 61%

“…In the first BT of our case, there were dilated hyperdense images at the left cortical vein at the vertex level, and there also were hyperdense hemorrhagic areas, which have peripheral hypodense edematous areas in the left parietal lobe. On the other hand, BT with contrast is a better modality for identifying dural venous sinus thrombosis, and the vascular endoluminal area at the site of the thrombosis is relatively hypodense (delta sign) (3,5,8,11). MR imaging and MR venography are the most sensitive techniques in determining CVST.…”

Section: Discussionmentioning

confidence: 99%

“…This entity can also result in severe mortality and morbidity if the diagnosis and treatment initiation are late. 12 adult cases with cerebral venous sinus thrombosis in nephrotic syndrome were reported in the literature (1)(2)(3)(5)(6)(7)(8)(9)(10)(11)(12)(13)(14)(15)(16). In this case, we are presenting an adult nephrotic syndrome patient with internal jugular venous thrombosis, in addition to intracranial segmental cerebral venous sinus thrombosis.…”

Section: Introductionmentioning

confidence: 94%

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Cerebral venous sinus thrombosis with internal jugular venous thrombosis in a male patient with nephrotic syndrome

Hacıfazlıoğlu

Arslan²,

Arslan³

et al. 2014

Turkish Neurosurgery

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Cerebral venous sinus thrombosis with internal jugular vein thrombosis is not reported as a complication in nephrotic syndrome. We report a 40-year-old male with nephrotic syndrome, who had headache during his hospitalization. Conventional diagnostic tests showed extensive thrombosis at the proximal part of superior sagittal sinus, left cortical vein, left sigmoid-transverse sinus and left internal jugular vein. The patient underwent medical treatment and was discharged in good health after 2 weeks. The aim of this study is to report a novel case of cerebral venous sinus thrombosis with internal jugular venous thrombosis in a male patient with nephrotic syndrome.

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Section: Discussionmentioning

confidence: 57%

Section: Discussionmentioning

confidence: 61%

Section: Discussionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 94%

See 2 more Smart Citations

Cerebral venous sinus thrombosis with internal jugular venous thrombosis in a male patient with nephrotic syndrome

Hacıfazlıoğlu

Arslan²,

Arslan³

et al. 2014

Turkish Neurosurgery

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“…38 It has been suggested that pseudotumor cerebri is due to undiagnosed CVT, and one study has suggested that MR venography should be done unless MR imaging is diagnostic for CVT in any patient with presumed IIH, to rule out this condition. Lin et al 34 found that 9.4% of patients (10 of 106) meeting criteria for IIH had thrombosis evident on MR venography. However, the classic demographic for IIH (a young, obese female) was not sensitive enough to allow for MR venography in just this subset, as the diagnosis in 4 patients would have been missed.…”

mentioning

confidence: 99%

From presentation to follow-up: diagnosis and treatment of cerebral venous thrombosis

Bentley¹,

Figueroa

Vender³

2009

FOC

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Cerebral venous thrombosis is an uncommon cause of stroke but remains a challenge for physicians faced with this diagnosis largely due to the variability in presentation. Anticoagulation, typically with intravenous heparin, remains the mainstay of treatment for stable patients and is sufficient in the majority of cases. However, a significant mortality rate exists for cerebral venous thrombosis due to patients who deteriorate or do not adequately respond to initial treatments. It is in these patients that more aggressive interventions must be undertaken. The neurosurgeon is often called on, either acutely for initial evaluation of the stroke or venous hemorrhage or after the failure of initial therapy for clot evacuation, hemicraniectomy, or thrombectomy. A proper workup must include a search for an underlying, correctable cause as well as thorough follow-up with correction of identified risk factors to decrease the risk of recurrent disease.

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Bilateral Papilledema and Partially Empty Sella in a Woman in Her 30s

Luo

Lü

2019

JAMA Ophthalmol

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A woman in her 30s was referred for evaluation of bilateral papilledema and progressive visual loss in the right eye, which deteriorated to blindness in 3 months. She denied any other neurologic symptoms, such as headache, nausea, vomiting, seizures, and paresis. She had a 4-year history of Behçet disease (BD), and her condition had been stabilized with thalidomide. Her optic neuropathy of BD had been treated with high-dose intravenous methylprednisolone for 4 days followed by oral prednisone for a month without improvement.Her best-corrected visual acuity was no light perception OD and 20/20 OS. Intraocular pressure was 21 mm Hg OD and 19 mm Hg OS. Fundus examination revealed papilledema in both eyes (Figure 1A). No obvious sign of optic disc ischemia or active uveitis was observed by fundus fluorescein angiography bilaterally. Humphrey visual fields showed complete visual field loss in the right eye and peripheral visual field defects in the left eye. Her anterior segment was normal in both eyes except for relative afferent pupillary defect in the right eye. Cranial nerve examination was otherwise unremarkable. Magnetic resonance imaging of the brain showed no remarkable abnormalities except for partially empty sella (Figure 1B). She had normal blood pressure levels. Results of laboratory testing regarding systemic infection were negative, and her serum D-dimer level was normal. Diagnosis Cerebral venous thrombosis What to Do Next C. Obtain cerebral magnetic resonance venography or computed tomographic venographyClinical Review & Education

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Occurrence of Cerebral Venous Sinus Thrombosis in Patients with Presumed Idiopathic Intracranial Hypertension

Cited by 94 publications

References 20 publications

Cerebral venous sinus thrombosis with internal jugular venous thrombosis in a male patient with nephrotic syndrome

Cerebral venous sinus thrombosis with internal jugular venous thrombosis in a male patient with nephrotic syndrome

From presentation to follow-up: diagnosis and treatment of cerebral venous thrombosis

Bilateral Papilledema and Partially Empty Sella in a Woman in Her 30s

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