Orbital Hemorrhage and Compressive Optic Neuropathy in Patients With Midfacial Fractures Receiving Low-Molecular Weight Heparin Therapy

Jamal, Basem T.; Diecidue, Robert J.; Taub, Daniel; Champion, Allen F.; Bilyk, Jurij R.

doi:10.1016/j.joms.2008.12.044

Cited by 20 publications

(9 citation statements)

References 14 publications

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“…In general, patients treated before the 2 h from the symptoms start achieve a final Snellen visual acuity of 6/12 or better, almost 15% reach a final outcome of less than 6/12 [2,. Outcomes in patients treated after more than 2 h are worst and almost 25% of them reach final visual acuity of 6/12 or better [5,15,20,23,24,28,29,[31][32][33][34][35][36]55]. In those cases, who reached a visual acuity of 6/12 or better even if treated after 2 h, it should be clarified that more than 50% of them presented a visual acuity of 6/12 or better at the admission.…”

Section: Orbital Compartment Syndromementioning

confidence: 95%

Timing of surgical intervention for compartment syndrome in different body region: systematic review of the literature

et al. 2020

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Compartment syndrome can occur in many body regions and may range from homeostasis asymptomatic alterations to severe, life-threatening conditions. Surgical intervention to decompress affected organs or area of the body is often the only effective treatment, although evidences to assess the best timing of intervention are lacking. Present paper systematically reviewed the literature stratifying timings according to the compartmental syndromes which may beneficiate from immediate, early, delayed, or prophylactic surgical decompression. Timing of decompression have been stratified into four categories: (1) immediate decompression for those compartmental syndromes whose missed therapy would rapidly lead to patient death or extreme disability, (2) early decompression with the time burden of 3–12 h and in any case before clinical signs of irreversible deterioration, (3) delayed decompression identified with decompression performed after 12 h or after signs of clinical deterioration has occurred, and (4) prophylactic decompression in those situations where high incidence of compartment syndrome is expected after a specific causative event.

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Section: Orbital Compartment Syndromementioning

confidence: 95%

Timing of surgical intervention for compartment syndrome in different body region: systematic review of the literature

et al. 2020

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“…Clinically, most patients treated within 2 hours will achieve a final Snellen visual acuity better than 6/12, though approximately 15% will be worse than 6/12 7,13,18,22,29,35,36,40,49,52,53,55–66. Patients treated after 2 hours have poorer reported outcomes with approximately 25% of patients reaching a final visual acuity of 6/12 or better 1,7,19,24,44,47,48,50,51,53,60,64,65,67–71. For those cases who achieve a favorable visual acuity despite being treated after 2 hrs, more than 50% had a presenting acuity of 6/12 or better.…”

Section: Managementmentioning

confidence: 99%

<p>Orbital Compartment Syndrome: An Update With Review Of The Literature</p>

McCallum¹,

Keren²,

Lapira³

et al. 2019

OPTH

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Orbital compartment syndrome (OCS) is a potentially blinding condition characterized by a rapid increase in intra-orbital pressure. OCS is most commonly seen in the context of intra-orbital hemorrhage secondary to either trauma or surgery. A review of the literature indicates that better visual outcomes are achieved when interventions occur within the first 2 hrs. There are reports of visual recovery after a delay in management and consideration should be given to intervention even when presentation is delayed. Reported interventions include: lateral canthotomy with cantholysis, bony orbital decompression and treatment of the underlying cause.

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“…22,23 However, drug therapy or hemorrhage due to congenital or systemic conditions may lead to nontraumatic RBH. [24][25][26][27] Drug-related nontraumatic RBH occurs in hemorrhagic diatheses due to circulating anticoagulation medication, whereas vessel malformations are congenital causes of RBH. [28][29][30] RBH can occur spontaneously in drug-related pharmacological or congenital conditions.…”

Section: Retrobulbar Hemorrhagementioning

confidence: 99%

Diagnosis and Treatment of Optic Nerve Trauma

et al. 2014

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Decreasing visual acuity secondary to orbital trauma or orbital and anterior skull base surgery may be caused by either sudden space-occupying intraorbital lesions, including retrobulbar hemorrhage (RBH), or direct damage to the prechiasmatic pathway. Contrary to traumatic optic neuropathy, RBH must be diagnosed and treated immediately to prevent permanent damage to the visual system. Therefore, monitoring and handling of visual pathway damage are mandatory. Flash visual evoked potentials and electroretinograms can provide evidence of the status of conductivity of the visual pathway when clinical assessment is not feasible. Both are thus essential diagnostic procedures not only for primary diagnosis but also for intraoperative evaluation. In case of RBH surgical decompression is compulsory. However, traumatic optic neuropathy does not respond to either corticosteroids or optic canal surgery. Modern craniomaxillofacial surgery requires detailed consideration of the diagnosis and treatment of traumatic visual pathway damage with the ultimate goal of preserving visual acuity.

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Orbital Hemorrhage and Compressive Optic Neuropathy in Patients With Midfacial Fractures Receiving Low-Molecular Weight Heparin Therapy

Cited by 20 publications

References 14 publications

Timing of surgical intervention for compartment syndrome in different body region: systematic review of the literature

Timing of surgical intervention for compartment syndrome in different body region: systematic review of the literature

<p>Orbital Compartment Syndrome: An Update With Review Of The Literature</p>

Diagnosis and Treatment of Optic Nerve Trauma

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