Original article Pityriasis rubra pilaris-an historical approach. 2 CLINICAL FEATURES

Griffiths, W.A.D.

doi:10.1111/j.1365-2230.1976.tb01397.x

Cited by 62 publications

(52 citation statements)

References 39 publications

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“…Although in a few cases the onset or exac erbation of PRP might have been related to a previous sunburn [7] or to sunlight [3,8,9], to our knowledge this is the first reported case exacerbated by UVB phototherapy. It seems that phototherapy (UVA or UVB) is best avoided in PRP.…”

Section: P Ityriasis Rubra Pilaris Exacerbated By U Ltraviolet B Photmentioning

confidence: 94%

Pityriasis rubra pilaris Exacerbated by Ultraviolet B Phototherapy

Yaniv¹,

Barzilai²,

Trau³

1994

Dermatology

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Section: P Ityriasis Rubra Pilaris Exacerbated By U Ltraviolet B Photmentioning

confidence: 94%

Pityriasis rubra pilaris Exacerbated by Ultraviolet B Phototherapy

Yaniv¹,

Barzilai²,

Trau³

1994

Dermatology

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“…The tentative diagnosis of non-bullous ichthyosiform 76 January 1983 '77 erythroderma made at infancy was no longer tenable as he grew older. The facial erythroderma, follicular prominence of the rash and palI!10-plantar thickening might have suggested atYPlc~1 juvenile pityriasis rubra pilaris but the naevoid pattern was not described by Griffiths (1976 …”

Section: Discussionmentioning

confidence: 99%

Generalized Inflammatory Epidermal Naevus

Wong

Wells

1983

J R Soc Med

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“…Type I, classical adult PRP, is the most common form where adults are afflicted. As Griffith describes, the elementary lesion is follicular hyperkeratosis (Griffiths, 1976). Progressive erythroderma develops due to follicular hyperkeratosis.…”

Section: Clinical Presentation and Classificationmentioning

confidence: 99%

“…It was first described by Alaudius Tarral in 1828, and Devergie named it "pityriasis pilaris" in 1856 (Griffiths, 1976;Fox et al, 1985). In 1889, Besnier renamed the disease to PRP (Griffiths, 1976). This rare skin disorder occurs in all races, affects both sexes equally, and has been reported world wide (Griffiths, 1980).…”

Section: Introductionmentioning

confidence: 99%

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A Review of Pityriasis Rubra Pilaris and Rheumatologic Associations

Chan

Liu

Naguwa

2004

Journal of Immunology Research

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Pityriasis rubra pilaris (PRP) is a rare group of hyperkeratotic, papulosquamous disease that can be acquired or inherited. There have been reported cases of rheumatologic associations, mainly arthritis and dermatomyositis. In this review article, we will explore the clinical presentation and classification, rheumatologic associations and treatment modalities of PRP. In addition, we will also report a case of PRP with seronegative arthritis.Keywords: Pityriasis rubra pilaris; Review; Arthritis; Rheumatologic; Treatment CASE REPORTA 50-year-old East Indian male with a 2-year history of nonspecific knee arthritis presented to his primary care doctor with a new scaly and pruritic rash. It began on the extensor surfaces of the elbow and bilaterally spread onto the forearms. Similar lesions began to appear over the scalp, trunk, axilla and knee. His nails became thickened and hyperpigmented. The patient was diagnosed with psoriasis. With the appearance of the lesions, his knee pain intensified, especially when ascending the stairs. The pain would awaken him at night. During this period, he had stiffening of his extremities. Besides fatigue, he denied any recent acute illnesses, constitutional symptoms, and mucocutaneous or ocular lesions. His past medical history included hypothyroidism and seasonal allergies. He denied food, drug and environmental allergies. His medication list at presentation included levothyroxine and glucosamine and chondroitin. He smoked tobacco occasionally and owned a cat. He had not traveled outside the country recently. His mother had nonspecific arthritis. On physical examination, his vitals were normal. There were no lymphadenopathy, oral lesions and hepatosplenomegaly. Neither joint deformity nor synovitis was appreciated. He had tenderness of the right knee on palpation with mild patellar laxity. He had multiple sharply demarcated redorange scaly plaques over the extensor surfaces of the forearms, axillae, knees and trunk. The plaques were nearly confluent on his trunk with the exception of islands of normal skin. The skin of his palms was thick and dry. Bilaterally, his second and third fingernails were hyperkeratotic with pitting and hyperpigmentation.X-rays of the hands and knees were normal. Complete blood count, complete metabolic panel and urinalysis were normal. The erythrocyte sedimentary rate was 15; antinuclear antibody was 1:160 with a nucleolar pattern. Thyroid stimulating hormone, immunoglobulin and complement levels were normal. Rheumatoid factor, hepatitis panel and RPR were negative. He did not respond to the conventional psoriatic treatment with ultraviolet light therapy UVA and UVB. Eventually, a punch biopsy was performed, and a diagnosis of pityriasis rubra pilaris (PRP) was made. The patient was placed on methotrexate therapy and his arthritis and skin lesions subsequently resolved.

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Original article Pityriasis rubra pilaris-an historical approach. 2 CLINICAL FEATURES

Cited by 62 publications

References 39 publications

Pityriasis rubra pilaris Exacerbated by Ultraviolet B Phototherapy

Pityriasis rubra pilaris Exacerbated by Ultraviolet B Phototherapy

Generalized Inflammatory Epidermal Naevus

A Review of Pityriasis Rubra Pilaris and Rheumatologic Associations

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