Encyclopedia of Life Sciences 2009
DOI: 10.1002/9780470015902.a0001376.pub2
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Oxidative Stress

Abstract: Respiring organisms derive multiple benefits from molecular oxygen. However, some fraction of the oxygen used is unavoidably converted to dangerous substances such as the superoxide radical (O 2 − ), hydrogen peroxide (H 2 O 2 ) and the hydroxyl radical (HO). These intermediates of oxygen reduction threaten the integrity of diverse biological molecules and hence the life of cells and organisms. The seeming comfort… Show more

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(1 citation statement)
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“…Cells also have antioxidant mechanisms to protect themselves from the burden of ROS, such as superoxide dismutase (SOD), glutathione peroxidase (GPx), and catalases [ 36 ]. Enhanced oxidative stress has been shown to be an exacerbating factor in pathologies including inflammation, reperfusion injuries, and ageing [ 37 ]. Uncoupling of eNOS in the endothelium may lead to oxidative stress and endothelial dysfunction via numerous mechanisms [ 38 ].…”
Section: Main Proatherogenic Risk Factors and Epigenetic-associatementioning
confidence: 99%
“…Cells also have antioxidant mechanisms to protect themselves from the burden of ROS, such as superoxide dismutase (SOD), glutathione peroxidase (GPx), and catalases [ 36 ]. Enhanced oxidative stress has been shown to be an exacerbating factor in pathologies including inflammation, reperfusion injuries, and ageing [ 37 ]. Uncoupling of eNOS in the endothelium may lead to oxidative stress and endothelial dysfunction via numerous mechanisms [ 38 ].…”
Section: Main Proatherogenic Risk Factors and Epigenetic-associatementioning
confidence: 99%