P-wave duration changes and dispersion in preeclampsia

Kırbaş, Özgür; Biberoğlu, Ebru; Kırbaş, Ayşe; Dağlar, Halil Korkut; Kurmuş, Özge; Uygur, Dilek; Danışman, Nuri

doi:10.1016/j.ejogrb.2014.10.042

Cited by 21 publications

(25 citation statements)

References 53 publications

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“…The diagnosis of severe PE was based on the presence of any of the following criteria: systolic blood pressure ≥160 mm Hg or diastolic blood pressure ≥110 mmHg on two separate measurements performed at sixhour intervals at the least, increased serum creatinine (>1.1 mg/ dL), headache, visual impairment, epigastric or right upper quadrant pain, elevated hepatic enzymes, thrombocytopenia (PLT<100000/mm 3 ), or pulmonary edema [1]. Pregnant with severe PE were divided into two groups: early-onset severe PE (<34 th week) (EOS-PE) and late-onset severe PE (≥34 weeks) (LOS-PE) [2]. Fetal growth restriction (FGR) was diagnosed when estimated fetal weight was below the 10th percentile, with abnormal fetal Doppler parameters.…”

Section: Methodsmentioning

confidence: 99%

“…Especially severe form of PE carries the high risks of disseminated intravascular coagulation; stroke, liver infarction and abruption of placentae, pulmonary edema, renal failure and thereby, may be cause of fetal and maternal morbidity and mortality [1,2]. PE can be classified on the timing of disease onset, as early-onset (EO), occurring before 34th gestational week and late-onset (LO) occurring 34th gestational week or later.…”

Section: Introductionmentioning

confidence: 99%

“…It is suggested that while EO-PE is primarily associated with defective placentation; LO-PE likely occurs due to a failure of maternal adaptation to pregnancy [3]. Endovascular invasion of the spiral arteries by extravillous trophoblast (EVT) is a crucial step for both initial implantation and subsequent placentation [1,2,5].…”

Section: Introductionmentioning

confidence: 99%

“…The etiology of the disease is heterogeneous and has not yet been identified. Multiple factors have been suggested in its pathophysiology, such as inflammation, defective placentation; cardiovascular maladaptation to pregnancy; malfunction in immunological, hormonal, genetic, and angiogenic mechanisms and the increased oxidative stress [1][2][3][4].…”

Section: Introductionmentioning

confidence: 99%

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Circulating ADAMTS-12 levels in early- and late-onset severe preeclampsia

Kırbaş¹,

Dağlar²,

Tımur³

et al. 2016

Med-Science

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Aim of this study is to determine whether maternal serum ADAMTS-12 levels differ among early-onset (<34 th week) severe preeclampsia (EOS-PE), late-onset (≥ 34 th week) severe preeclampsia (LOS-PE), and uncomplicated pregnancies. In this case-control study, maternal serum samples obtained from 22 pregnant patients with EOS-PE, 25 pregnant patients with LOS-PE, and 35 healthy patients with uncomplicated pregnancies (control group). The distinctly higher rate of negative perinatal outcomes in both EOS-PE and LOS-PE patients were recorded. There were no significant differences among the ADAMTS-12 levels of the groups. The main questions that need to be answered are whether the only difference between these two diseases is the time of their onset and whether the only difference between them with respect to fetal morbidity and mortality is prematurity.

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Section: Methodsmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

See 2 more Smart Citations

Circulating ADAMTS-12 levels in early- and late-onset severe preeclampsia

Kırbaş¹,

Dağlar²,

Tımur³

et al. 2016

Med-Science

Self Cite

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“…22 Hoogsteder et al studied ECG changes among hypertensive pregnant women and followed up the presence or severity of preeclampsia in the next pregnancy. They found that those who developed preeclampsia earlier in the index pregnancy or those who had recurrent preeclampsia in the subsequent pregnancy; had higher rate of ECG findings compared to those who had later onset of preeclampsia in the index pregnancy or those who had uneventful subsequent pregnancy.…”

Section: Ecg Abnormalities Among Preeclamptics and Future Cardiovascumentioning

confidence: 99%

Electrocardiographic profile of patients with hypertensive disorders in pregnancy

Desai

Prasad

2017

Int J Reprod Contracept Obstet Gynecol

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Background: Preeclampsia in pregnancy is a frequently encountered entity. Recent evidence suggests that preeclampsia can be considered as a screening test for future cardiovascular problems and abnormalities in pregnancy may help in counselling patients regarding future risk of cardiovascular problems. Electrocardiography (ECG) is a simple tool for detecting cardiovascular problems. While physiological changes in the ECG in pregnancy have been documented, the pattern of ECG in hypertensive disorders of pregnancy is not adequately studied in India. This formed the basis of the study.Methods: 140 patients who have been diagnosed as gestational hypertension and pre-eclampsia during the pregnancy were studied in the antenatal period. An ECG (non-portable, standard, 12 lead) available in the hospital was done, and the ECG was reported by the physician on-call.Results: The average age distribution of the patients was 28.26 years. Majority of the patients in the study were primigravidae. 35% of the patients were more than 37 weeks of gestation. 20 patients showed electrocardiographic abnormalities. The most common abnormality was sinus tachycardia and left axis deviation.Conclusions: ECG abnormalities are seen in 14.2% of preeclamptic women. ECG abnormalities in preeclamptic women studied had no relation to the maternal age, gestational age; parity and use of antihypertensives.

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