Parathyroid activity in hyperthyroidism

Bommer, Jürgen; Ritz, Eberhard; Gengenbach, E.

doi:10.1007/bf02546464

Cited by 8 publications

(2 citation statements)

References 5 publications

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“…[ 18 ] The above observation has been confirmed by other investigators as well. [ 19 ] However, some researchers could not demonstrate a decline in serum PTH levels among patients with hyperthyroidism, possibly due to difficulty in differentiating between normal and decreased concentrations due to less sensitive PTH assays. There is inverse relationship between serum calcium and serum PTH levels, indicating that increased serum calcium levels inhibit PTH secretion from parathyroid gland.…”

Section: P Arathyroid H Ormone mentioning

confidence: 99%

Thyroid disorders and bone mineral metabolism

Dhanwal

2011

Indian J Endocr Metab

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Thyroid diseases have widespread systemic manifestations including their effect on bone metabolism. On one hand, the effects of thyrotoxicosis including subclinical disease have received wide attention from researchers over the last century as it an important cause of secondary osteoporosis. On the other hand, hypothyroidism has received lesser attention as its effect on bone mineral metabolism is minimal. Therefore, this review will primarily focus on thyrotoxicosis and its impact on bone mineral metabolism.

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Section: P Arathyroid H Ormone mentioning

confidence: 99%

Thyroid disorders and bone mineral metabolism

Dhanwal

2011

Indian J Endocr Metab

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“…All groups had an increase in urinary excretion of phosphate in contrast to what would be expected, if the decrease in S‐PTH was of physiological importance. The increase in U‐phosphate is most likely a direct effect of thyroid hormones on the kidneys; however, efflux of phosphate from soft tissues and the bone after stimulation of resorptive activity is also a possible mechanism behind the increased renal excretion of phosphate 66…”

Section: Discussionmentioning

confidence: 99%

Is Skeletal Responsiveness to Thyroid Hormone Altered in Primary Osteoporosis or Following Estrogen Replacement Therapy?

Langdahl

Loft

Møller

et al. 1997

Journal of Bone and Mineral Research

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Hyperthyroidism is characterized by increased bone turnover and resorptive activity. Similar changes in remodeling are seen in osteoporosis. To study the pathogenetic role of thyroid hormone in osteoporosis, we measured concentrations of free and total thyroid hormones and investigated the sensitivity of the skeleton toward thyroid hormones in 14 osteoporotic, 16 estrogen-treated, and 15 normal postmenopausal women with comparable thyroid status. Triiodothyronine (T 3 , 60 g/day for 7 days) was administered to the three groups. The skeletal response was assessed by monitoring bone alkaline phosphatase (

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