Postirradiation survival kinetics of mammalian cells grown in culture

Elkind, M. M.; Sutton, Harriet; Moses, W. B.

doi:10.1002/jcp.1030580412

Cited by 91 publications

(15 citation statements)

References 7 publications

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“…Finally, after about 40 hours, survival and multiplicity increase at the same rate . This similarity in rates, originally reported by Elkind, Sutton and Moses (1961), indicates that cells within a group survive independently and transfer neither protective nor lethal substances to adjacent cells . Essentially the same conclusions can be reached for treatment, with hyperthermia, either administered alone or combined with X-irradiation, with the exception that the minimum in survival at about 6-8 hours was not observed (figures 2 and 3 and data not shown) .…”

Section: Discussionsupporting

confidence: 63%

Changes in Cellular Heat and/or Radiation Sensitivity Observed at Various Times after Trypsinization and Plating

Raaphorst

Sapareto

Freeman

et al. 1979

International Journal of Radiation Biology and Related Studies

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Section: Discussionsupporting

confidence: 63%

Changes in Cellular Heat and/or Radiation Sensitivity Observed at Various Times after Trypsinization and Plating

Raaphorst

Sapareto

Freeman

et al. 1979

International Journal of Radiation Biology and Related Studies

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“…(2) Although ‡™Dn/f(NS) decreases with n in the cases of 1,500 R and 2,000 R, there is a maximum of ‡™Dnf(NS) at n=3 in the case of 1,000 R.…”

Section: )mentioning

confidence: 92%

“…We determined the number of cell devision at various time after irradiation using equation (2). As shown in Figs.…”

Section: )mentioning

confidence: 99%

The Distribution of the Number of Postirradiation Cell Division

Sasaki¹

1969

Tohoku J. Exp. Med.

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“…In the first case, the ''target theory'' (TimofeefRessovsky and Zimmer 1947) suggested that even extremely weak ionizing radiations can lead the cell to die, as expected from the fact that in real experimental conditions the function f = e D/D [1 -(e DD2 )] N , which describes the development of cell survival vs. the intensity of ionizing radiation (Elkind et al 1961), never parallels the abscissa, measured in Gy, starting from its initial ''shoulder'' (Volpe 1999).…”

Section: Resultsmentioning

confidence: 98%

Mechanisms of the target response to magnetic fields and their correlation with the biological complexity

Volpe

Eremenko

2007

Environmentalist

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The present contribution points to the mechanisms of bioresponse caused by magnetic fields, paying attention to their action not only at the ionic, molecular or macromolecular levels but also at the levels of cells, tissues and organisms. The significance of findings concerning the magnetic-field dependence of cell proliferation, necrosis or apoptosis and cell volume or membrane fluidity is judged by comparing the results obtained in a solenoid, where a magnetic field can be added to the geomagnetic field, with those obtained in a magnetically shielded room, where the magnetic fields can be attenuated or null. This comparative criterion was particularly required when the differences found between the data provided by experimental samples and the data provided by control samples are small per se, as observed in estimating the magnetic-field dependence of expression of single genes or the magnetic-field dependence of total genome replication, transcription and translation. The report analyzes the magnetic-field dependence of the interactions between host animal cells and infecting bacteria and, in the framework of studies on the origin and adaptation of life on Earth, theoretical insights paving the way to elucidating the mechanisms of magnetic-field interactions with biosystems of different orders of organization are considered from the viewpoint of the ''biological windows'' thermodynamics. Thus, analogously to what is known for ionizing radiations, an inverse correlation emerged between the intensity of given magnetic fields and the biological complexity.

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Postirradiation survival kinetics of mammalian cells grown in culture

Cited by 91 publications

References 7 publications

Changes in Cellular Heat and/or Radiation Sensitivity Observed at Various Times after Trypsinization and Plating

Changes in Cellular Heat and/or Radiation Sensitivity Observed at Various Times after Trypsinization and Plating

The Distribution of the Number of Postirradiation Cell Division

Mechanisms of the target response to magnetic fields and their correlation with the biological complexity

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