Prevalence and Etiologic Factors of Non Carious Cervical Lesions among Prison's Population in Dakar

Abstract

To understand the role of stress in the occurrence of noncarious cervical lesions. Preventive measures may be instituted if the causative factors are detected and their modifying factors are considered. Clinical SignificanceBackground: The last two decades have provided a plethora of new studies concerning noncarious cervical lesions. Most significant studies are reviewed and integrated into a practical approach to the understanding and designation of abrasion, abfraction and erosion.

Features of Dentine Chemical Composition of Intact Teeth and Teeth With Wedge-Shaped Defects

“…Epidemiological studies of non-carious cervical lesions (NCCL) are carried out in all regions of the world that is fully justified by their high prevalence (36.8% [1], 52% [2], 67.8% [3], 68.5% [4], 88.1% [5]) and possible consequences [4]. The results of such studies are very important for identifying new etiological factors taking into account the patient's individual characteristics that are responsible for different degrees of the loss of dental hard tissue and the development of combined forms of damage [4,6].…”

“…The most likely causes of NCCL development are abrasion (61.60%), erosion (26.08%), abfraction (12.32%) [1] or any combination of them [2,6]. The frequency of this pathology increases with age [3][4][5][6] and the number of retained teeth [6] that indicates the long-term component in their formation associated with occlusive stress [1,3,5] which initially develops in enamel cracks [6,7].…”

“…The most likely causes of NCCL development are abrasion (61.60%), erosion (26.08%), abfraction (12.32%) [1] or any combination of them [2,6]. The frequency of this pathology increases with age [3][4][5][6] and the number of retained teeth [6] that indicates the long-term component in their formation associated with occlusive stress [1,3,5] which initially develops in enamel cracks [6,7]. The presence of microcracks leads to disruption of chemical bonds between hydroxyappatite crystals, subsequent penetration of water and other molecules making a tooth more vulnerable to dissolution, chemical erosion or abrasive factors [7].…”