Prognostic significance of electrodiagnostic studies in the Guillain-Barre syndrome.

Peripheral nerve conduction in Miller Fisher syndrome.

“…It has been suggested that evidence of denervation in the Guillain-Barre syndrome may indicate that clinical recovery will not be complete (McLeod et al, 1976;Raman and Taori, 1976). Fibrillation in the facial muscles in three cases of Miller Fisher syndrome was claimed by Van Allen and Mac-Queen (cases 2, 3, 4) but were not reported in our case 2.…”

A quantitative electrophysiological study of acute idiopathic polyneuritis.

“…An extensive literature on this condition has yielded largely qualitative information on the pathological concomitants of the disease (Peterman et al, 1959;Bannister and Sears, 1962;Humphrey, 1964;lsch et al, 1964;Lambert and Mulder, 1964;Bergamini et al, 1966;McQuillen, 1971; Kimura and Butzer, 1975;McLeod et al, 1976;Raman and Taori, 1976). The introduction of quantitative methods of electromyography (McComas et al, 1971; Ballantyne and Hansen, 1974a, b) has contributed new information on the neurophysiological changes in a number of neuromyopathies Hansen, 1974a, b, 1975).…”

Diabetes mellitus may affect short‐term outcome of Guillain‐Barré syndrome

“…Previous studies identified different predictors of a poor outcome in GBS, with the following being the most common: older age, clinical severity, mechanical ventilation, absent deep tendon reflexes, longer duration of hospitalization and of nadir, longer time from weakness until beginning of improvement, preceding diarrhea, dysautonomia, higher protein level in cerebrospinal fluid, lower serum sodium level, axonal features in nerve conduction studies, and denervation on needle electromyography (Eberle et al, ; Raman and Taori, ; Winer et al, , ; Rajabally and Uncini, ; Kalita et al, ; Sipilä et al, ) . Identified predictors of short‐term prognosis in GBS are: age, absence of antecedent infection, time from onset to admission, facial, glossopharyngeal and vagal nerve deficits, weakness at admission and nadir, lack of foot flexion after immunotherapy, mechanical ventilation, and sciatic motor conduction block (Fourrier et al, ; González‐Suárez et al, ; Wu et al, ) .…”

“…Guillain‐Barré syndrome (GBS) is an acute autoimmune disorder of the peripheral nerves and their roots (Willison et al, ) . Multiple factors have been shown as important for prognosis of GBS: age, preceding diarrhea, degree of weakness, mechanical ventilation, duration of nadir, duration of hospitalization, deep tendon reflexes, dysautonomia, protein level in cerebrospinal fluid, serum sodium level, findings on nerve conduction studies, and treatment (Eberle et al, ; Raman and Taori, ; Winer et al, , ; van Koningsveld et al, ; Rajabally and Uncini, ; Kalita et al, ; Sipilä et al, ) . The Erasmus GBS Respiratory Insufficiency Score for short‐term prognosis of GBS comprises the strongest predictors – time between onset and admission, the presence of facial and/or bulbar weakness, and the degree of weakness on admission (Walgaard et al, ) .…”