Pseudo-Atopic Dermatitis

Shanon, J

doi:10.1159/000254470

Cited by 28 publications

(4 citation statements)

References 8 publications

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confidence: 99%

“…12–15 The flare-up of healing or quiescent contact dermatitis is the most frequent reaction, but pompholix, dyshidrosic or vescicular eczema, toxicoderma-like rash, chronic pruritus, maculo-papular rash, vasculitis-like lesions, flexural dermatitis, papuloerythroderma-like eruptions, chronic urticaria, pseudo-atopic dermatitis, generalized rash, and baboon syndrome are several clinical variants within the spectrum of systemically induced allergic contact dermatitis (ACD). 1–21 SCD should be considered as an “umbrella term,” which includes a broad spectrum of clinical manifestations that can occur when a person sensitized to a contact allergen is exposed to the same allergen through a systemic route.…”

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confidence: 99%

“…A small number of clinical and experimental studies suggest that dietary nickel could be the possible cause for the provocation and persistence of symptoms in some patients with concomitant nickel sensitization. [1][2][3][4][5][6][7][8][9][10][11] Nickel-sensitized patients, when exposed to the hapten given per os, can develop a flare-up reaction of previous contact dermatitis. Systemic contact dermatitis (SCD) is the term normally used to describe clinical manifestations of the systemic provocation by nickel and some other haptens.…”

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confidence: 99%

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Nickel Sensitization and Dietary Nickel are a Substantial Cause of Symptoms Provocation in Patients with Chronic Allergic-Like Dermatitis Syndromes

Antico¹,

Soana²

2015

Allergy�Rhinol (Providence)

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Data in literature seem to show that, in patients with contact allergic dermatitis, dietary nickel might be a cause of systemic dermatitis, but little information exists in literature about the role of nickel sensitization and dietary nickel in patients with allergic-like chronic dermatitis syndromes. The prevalence of nickel sensitization in patients with chronic allergic-like, non-IgE-mediated skin diseases, and the possible impact of dietary nickel on symptom provocation and persistence has been assessed in the present retrospective study on a case series of 1726 patients referred to our allergy unit for chronic allergic-like skin diseases. IgE-mediated pathogenesis and other differential diagnoses excluded, patients were patch tested. Nickel-positive patients underwent an elimination diet and double-blind placebo-controlled nickel challenge (DBPCNC) test. A total of 339 (20%) tested nickel-positive. Fifty-two patients (15%) recovered by avoiding sources of nickel contact and 29 (10%) dropped out. Out of the remaining nickel-sensitized patients, 277 (80%) achieved complete or near complete recovery with low-nickel content diet, and 185 of them (89%) were positive to DBPCNC. We conclude that nickel sensitization and dietary nickel seem to be the chief trigger for provocation and persistence of symptoms in an important part (∼11%) of patients with chronic allergic-like dermatitis syndromes.

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confidence: 99%

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confidence: 99%

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Nickel Sensitization and Dietary Nickel are a Substantial Cause of Symptoms Provocation in Patients with Chronic Allergic-Like Dermatitis Syndromes

Antico¹,

Soana²

2015

Allergy�Rhinol (Providence)

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“…The term "Baboon syndrome" was used firstly in 1984 by Andersen et al to describe a generalized dermatitis of the buttocks, anogenital area, flexures, and eyelids, frequently observed in patients with systemic contact dermatitis to nickel [8] as Shannon pseudo atopic dermatitis was firstly identified by Shannon as a variant of flexural eczema resembling an atopic dermatitis induced by chrome, but it can be elicited by Ni too [9].…”

Section: Foods Containing High Level Of Nimentioning

confidence: 99%

Nickel Hypersensitivity: A General Review on Clinical Aspects and Potential Co-Morbidities

GF¹,

Bonamonte²

2016

J Allergy Ther

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Allergic contact hypersensitivity to metals is a delayed-type allergy. Although various metals are able to induce an allergic reaction, nickel is the most frequent cause of metal allergy worldwide. The high prevalence of skin sensitization to that metal is the result of an ubiquitous exposure, being contained in different everyday things, such as detergents, cheap jewelry, cosmetics, coins, buttons, zippers, eyeglasses, buckles, clasps, inks, dental prosthesis and foods even. However, nickel is able to induce a wide range of morphological and clinical patterns, ranging from simple allergic contact dermatitis to systemic contact dermatitis and systemic nickel allergy syndrome. The latter seems to be promoted by nickel rich-foods or through endogenous route caused by metal implants which seem to elicit mainly in loco symptoms, sometimes with catastrophic consequences as aseptic bone necrosis following the implantation of hip prosthesis.

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Carrier Specificity of Tuberculin-Type Reaction to Trivalent Chromium

Cohen¹

1966

Arch Dermatol

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Pseudo-Atopic Dermatitis

Cited by 28 publications

References 8 publications

Nickel Sensitization and Dietary Nickel are a Substantial Cause of Symptoms Provocation in Patients with Chronic Allergic-Like Dermatitis Syndromes

Nickel Sensitization and Dietary Nickel are a Substantial Cause of Symptoms Provocation in Patients with Chronic Allergic-Like Dermatitis Syndromes

Nickel Hypersensitivity: A General Review on Clinical Aspects and Potential Co-Morbidities

Carrier Specificity of Tuberculin-Type Reaction to Trivalent Chromium

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