Renal Artery Embolectomy

Foley, William J.; Kraft, Richard O.

doi:10.1001/archsurg.1971.01350120112021

Cited by 15 publications

(3 citation statements)

References 22 publications

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“…Clot on prosthetic valve and vulvular vegetations in bacterial endocarditis are also cited as potential sources of renal emboli. 10 One patient in the present series was also diagnosed with subacute bacterial endocarditis. Therefore, renal infarction resulted from cardiac-original was suspected as the most common source of embolization in the present series.…”

Section: Discussionmentioning

confidence: 63%

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Clinical characteristics of patients with segmental renal infarction

et al. 2006

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SUMMARY:Background: Renal infarction is usually an underestimated disease due to its rare and non-specific presentations; the renal survival of these patients is not well studied. The aim of the present analysis is to study the clinical features and outcome in patients who had documented renal infarction. Methods: Twenty-two patients (12 men and 10 women, mean age of 57.7 ± 3.44 years (28.4-83.3 years)) with image-confirmed segmental renal infarction in the past 15 years were enrolled. All patients were followed up at outpatient department with a median of 4 years (1-14 years). Initial and follow-up clinical characteristics and laboratory results were recorded. Results: The most common underlying disease was cardiovascular disease. Renal infarction often presented with non-specific symptoms, including flank pain (55%), vague abdominal pain (50%), nausea/vomiting (46%) and fever (27%). The levels of leucocytes, lactate dehydrogenase, blood urea nitrogen and serum creatinine were all elevated at admission. The early diagnosis group (12/22) had more obvious flank pain, nausea/vomiting (P < 0.001) and higher alanine transaminase (P = 0.02). It also predisposed to undergo antiplatelet or anticoagulant therapy (all P < 0.04). During follow up, there was no recurrence in the whole study group, and a trend of better recovery of renal function was noted in the early diagnosis group. Conclusion: The serum creatinine level correlates with longer hospitalization length (P < 0.05). As regards long-term prognosis, no definite factor or treatment was found to have significant effect in segmental renal infarction patients. However, early diagnosis and early initiation of treatment seems to have a positive effect on future renal outcome.

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Section: Discussionmentioning

confidence: 63%

“…And the authors also observed atrial fibrillation as the largest group (50%) in the present series. Clot on prosthetic valve and vulvular vegetations in bacterial endocarditis are also cited as potential sources of renal emboli 10 . One patient in the present series was also diagnosed with subacute bacterial endocarditis.…”

Section: Discussionmentioning

confidence: 65%

Clinical characteristics of patients with segmental renal infarction

et al. 2006

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“…10 A clot on a prosthetic valve and valvular vegetations in bacterial endocarditis are also cited as sources of renal emboli. 11 In addition to emboli of cardiac origin, hypercoagulability states like antiphospholipid syndrome and polycythemia vera are major risk factors for renal infarction. 12 It is rarely associated with a hypercoagulable state, although a microvascular state is more common.…”

Section: Treatmentmentioning

confidence: 99%

Renal infarction

Saeed¹

2012

IJNRD

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Abstract:Renal infarction is an underdiagnosed and under-reported phenomenon, and needs to be diagnosed rapidly to prevent permanent loss of renal function. Renal infarction should be considered in the initial differential diagnosis of nephrolithiasis and pyelonephritis. It is often mistaken for more benign pathology and is worthwhile reviewing and reporting. Keywords: renal infarction, diagnosis, atrial fibrillation Case reportA 77-year-old woman was admitted with severe epigastric pain radiating to the back, bloody diarrhea, and vomiting, which started a night prior to admission. She described her abdominal pain as being similar to cholecystitis pain which she had suffered many years ago. She had a history of chronic atrial fibrillation and was off warfarin pending eye surgery. There was no history of recent travel, nonsteroidal anti-inflammatory drug use, or any dietary changes. She denied any history of alcohol or recreational drug use. Her other relevant past medical history included a stroke in 1994, type 2 diabetes, hypertension, dyslipidemia, gastroesophageal reflux disease, and a history of stomach ulcers with gastrointestinal bleeding (diffuse hemorrhagic gastritis with Helicobacter pylori in 1999). Medications on admission included hydrochlorothiazide, metformin, gliclazide, amlodipine, digoxin, candesartan, metoprolol, spironolactone, atorvastatin, and eye drops.Physical examination revealed a blood pressure of 189/94 mmHg and a heart rate of 85 beats per minute, irregular. She was afebrile, with normal saturation. She had diffuse abdominal tenderness, but no rebound tenderness or guarding. She had melena and a hemoccult test was positive.Laboratory studies were done on admission, and showed amylase 365 (20-160) U/L, lipase 221 (8-78) U/L, white blood count 28.9 (4.8-10.8) × 10 9 /L, absolute neutrophil count 27.5 (2-7.5) × 10 9 /L, and lactate dehydrogenase 485 (115-220) U/L (Table 1). Electrolytes, renal function, and liver function were normal. Albumin was 43 (32-45) g/L and INR was 1.25 (0.8-1.20). The digoxin level was 0.79 (0.64-2.56) nmol/L. Urine analysis was unremarkable except for 2+ glucose, positive occult blood, and 10-20 red blood cells per high powered field in the urine. A 24-hour urinary protein was 0.19 (0.0-0.15) g/day and creatinine clearance was 67 mL per minute, with a daily volume of 2750 mL.On admission, a series of abdominal x-rays and a computed tomography (CT) scan were ordered. The abdominal-rays showed a nonspecific gas pattern. No fluid level was noted. A chest x-ray showed cardiomegaly with calcification and unfolding of the thoracic aorta. Abdominal CT showed no signs of pancreatitis or stones. An 8 mm focal area of infarction was noted in the left kidney as well as a small focal infarction in the right kidney, which indicated extensive atherosclerosis of the superior mesenteric artery. No evidence of ischemic bowel or perforation was noted. There were scattered diverticuli but no signs of diverticulitis. Air space disease was noted at the left lung base.A hypercoagula...

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Treatment of Renal Artery Embolism

Nicholas¹,

DeMuth²

1984

Arch Surg

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Renal Artery Embolectomy

Cited by 15 publications

References 22 publications

Clinical characteristics of patients with segmental renal infarction

Clinical characteristics of patients with segmental renal infarction

Renal infarction

Treatment of Renal Artery Embolism

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