Research on characterization and modeling for ultraviolet degradation of imidacloprid based on absorbance change

Wang, Xiaoyan; Ji, Rendong; Zhang, Yulin; Yang, Yudong; Fu, Chengfang; Yang, Dingli

doi:10.1016/j.ijleo.2017.09.110

Cited by 5 publications

(2 citation statements)

References 9 publications

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“…For instance, it was gradually discovered that imidacloprid is toxic to humans, and that imidacloprid is lethal to non-target organisms such as bats, earthworms, bees, fish, and shrimp (Table 1) [17][18][19][20][21]. Inhibits thyroid hormone secretion, causes abnormal alignment of the spermatogenic epithelium, epididymal hyperplasia, and oligospermia in male lizards [28][29][30] 7. Brown shrimp 0.5 g•L −1 Sublethal effect, especially in dehulling delay and growth reduction [20,31] 8.…”

Section: Toxicity Of Imidaclopridmentioning

confidence: 99%

“…After 14 days of imidacloprid exposure, significant destruction of earthworm epidermis and midgut tissues has been observed [21]. Meanwhile, imidacloprid and its metabolites are eliminated through the skin and excretion in Chinese lizards (Eremias argus) [28]. However, imidacloprid exposure inhibits thyroid hormone secretion in lizards, resulting in the down-regulation of thyroid hormone receptors and their associated metabolic enzyme genes [29].…”

Section: Toxicity To Humans and Terrestrial Animalsmentioning

confidence: 99%

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Insights into the Toxicity and Degradation Mechanisms of Imidacloprid Via Physicochemical and Microbial Approaches

Pang

Lin

Zhang

et al. 2020

Toxics

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Imidacloprid is a neonicotinoid insecticide that has been widely used to control insect pests in agricultural fields for decades. It shows insecticidal activity mainly by blocking the normal conduction of the central nervous system in insects. However, in recent years, imidacloprid has been reported to be an emerging contaminant in all parts of the world, and has different toxic effects on a variety of non-target organisms, including human beings, due to its large-scale use. Hence, the removal of imidacloprid from the ecosystem has received widespread attention. Different remediation approaches have been studied to eliminate imidacloprid residues from the environment, such as oxidation, hydrolysis, adsorption, ultrasound, illumination, and biodegradation. In nature, microbial degradation is one of the most important processes controlling the fate of and transformation from imidacloprid use, and from an environmental point of view, it is the most promising means, as it is the most effective, least hazardous, and most environmentally friendly. To date, several imidacloprid-degrading microbes, including Bacillus, Pseudoxanthomonas, Mycobacterium, Rhizobium, Rhodococcus, and Stenotrophomonas, have been characterized for biodegradation. In addition, previous studies have found that many insects and microorganisms have developed resistance genes to and degradation enzymes of imidacloprid. Furthermore, the metabolites and degradation pathways of imidacloprid have been reported. However, reviews of the toxicity and degradation mechanisms of imidacloprid are rare. In this review, the toxicity and degradation mechanisms of imidacloprid are summarized in order to provide a theoretical and practical basis for the remediation of imidacloprid-contaminated environments.

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