“…The two major etiologies of RHT are embolic (due to propagation of a DVT) or in situ (due to stagnant blood flow, as seen in cardiomyopathy and in arrhythmia, such as in atrial fibrillation). However, there are case reports documenting RHT secondary to pacemaker wires [ 38 ], central venous catheters [ 39 ], hemodialysis catheters [ 40 ], and embryological remnants [ 41 ], as a complication of ablation [ 42 ], formed during cardiac arrest with cardiopulmonary resuscitation [ 43 ], and after carbon monoxide poisoning [ 44 ]. With regard to our patient, it is likely that the RHT development was multifactorial.…”