Samuel Butler and human long term memory: Is the cupboard bare?

Case Study: The Impact of Holographic Kinetics on Heroin Addiction and “Criminal” Behaviors

“…Similarly, a cause can have an effect outside its front (future) light cone [22][23][24][25]. The action reaction pendulum of cause and effect feeds an invisible dimension within the internal world of a human energy field, creating a visible imbalance (anxiety, depression, etc.,) which needs to be brought back to null point zero vector observer within man's awareness by waking up to his responsibility of the metaphysical consequences of his own creation upon its own separate dimension of time [26,27].…”

Holographic Kinetics for Clinically Diagnosed Suicidal, Depressed, PTSD Military Veterans: Case Study

“…Consequently, a piece of a long-term memory is equally distributed over a dendritic arbor so that each part of the dendritic network contains all the information stored over the entire network of the genetic hereditary lines of the mother and father or beyond (epigenetics) and/or long term experience memories attached to the life force of the patient, coming through time [23][24][25][26][27][28][29][30][31].…”

Long-term memory: scaling of information to brain size

“…We can, however, speak about the size of the metaphorical “cupboard” where, conventional wisdom holds, long-term memory must lie (Draaisma, 2000). And if the cupboard proves to be bare, we have to admit a problem (Forsdyke, 2009). …”

“…Unfortunately, microcephalics tend to be tested more for intelligence (usually impaired), than for their ability to recall information. However, on the assumption that intelligence and long-term memory are to some extent related, we can note that a few microcephalics have normal intelligence (Forsdyke, 2009). Thus, with both savants and microcephalics, the evidence, albeit weak, suggests a disconnect between the volume of neural tissue held within the cranium and the quantity of information which that neural tissue is, in some way, held to store.…”

“…These plasticity explanations imply that, in keeping with the sometimes amazing recoveries reported for severe brain injuries, an otherwise-occupied part of the brain can change to compensate for a defective part. Under the prevailing neural network “connectionist” paradigm (Draaisma, 2000; Forsdyke, 2009), for hydrocephalics with only 5% of neural tissue remaining, this would seem to require the establishment early in life of a critical network, which would have to retain its connectivity in the face of the subsequent severe progressive distortion associated with ventricular expansion. Unfortunately, there are no post-mortem histological studies on this.…”