“…Lipid profi les in ARVD patients follow the same pattern as in patients with coronary or carotid atheroma, with signifi cantly reduced apolipoprotein A1 levels [ 47 ]. Other studies have shown increased levels of free-fatty acids (glycerol-glyceride) in patients with ARVD [ 48 ], though this may have more important implications for mortality than development of atheroma [ 49 ]. However, intervention with statins has been shown to prevent anatomical progression of RAS in a retrospective study, which provides some support to the pathogenic effect of hypercholesterolemia in renal atherogenesis [ 50 ].…”