“…Following curative surgical treatment,^'^ catheter ablation for AV nodal reentrant tachycardia was introduced in 1986 using DC shocks applied to the retrograde fast pathway, a few millimeters from the His hunjjlg 24.2.5 purther attempts to consistently ablate the "guilty" retrograde fast pathway without modifying anterograde conduction have been unsuccessful so that the risk of AV block targeting the fast pathway remains about 3% with the use of radiofrequency (RF) current.^'*"^'^ This risk is greater than using RF ahlation to target the "slow pathway" (SP) in which energy is applied 10-20 mm posterior to the His bundle with an incidence =1% of AV hlock. This approach Introduced by Jackman et al^° has become the therapeutic procedure of choice.^""""^^ Although ablation for AVJRT is dichotomized into fast or slow pathway ablation, there are arguments suggesting disruption of the link between botb pathways (or of an "intermediate" pathway) as the mechanism of ,U^,g 46,48,49 Different approaches to target the slow pathway have heen described. In the electrophysiological approach, electrogram patterns are nsed to identify the ablation site^"*^^ whereas in the anatomical approach, the ablation site is mainly selected on the basis of anatomical criteria (in combination with an atrial/ventricular electrogram ratio of 0.5 or less).…”