Silicone oil induced glaucoma: A review

Ichhpujani, Parul; Jindal, A.; Katz, L. Jay

doi:10.1007/s00417-009-1155-x

Cited by 170 publications

(154 citation statements)

References 80 publications

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“…34 The IOP elevation may range from mild and transient to severe and sustained. The early IOP spikes may be due to pupillary block, mechanical-angle obstruction by oil, or inflammation and the late-onset spikes are proposed to be due to synechial-angle closure, rubeosis iridis, oil droplet emulsification, and migration of silicone oil into the anterior chamber and clogging the outflow.…”

Section: Silicone Oilmentioning

confidence: 99%

Non-steroidal drug-induced glaucoma

Razeghinejad

Pro

Katz

2011

Eye

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Numerous systemically used drugs are involved in drug-induced glaucoma. Most reported cases of non-steroidal drug-induced glaucoma are closed-angle glaucoma (CAG). Indeed, many routinely used drugs that have sympathomimetic or parasympatholytic properties can cause pupillary block CAG in individuals with narrow iridocorneal angle. The resulting acute glaucoma occurs much more commonly unilaterally and only rarely bilaterally. CAG secondary to sulfa drugs is a bilateral non-pupillary block type and is due to forward movement of iris-lens diaphragm, which occurs in individuals with narrow or open iridocorneal angle. A few agents, including antineoplastics, may induce open-angle glaucoma. In conclusion, the majority of cases with glaucoma secondary to non-steroidal medications are of the pupillary block closed-angle type and preventable if the at-risk patients are recognized and treated prophylactically.

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Section: Silicone Oilmentioning

confidence: 99%

Non-steroidal drug-induced glaucoma

Razeghinejad

Pro

Katz

2011

Eye

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“…SO may cause some complications like secondary glaucoma and keratopathy by generating oil droplets due to the emulsification. 10 Raised IOP remains a significant complication of intraocular SO with reported incidences of between 6 and 40% of eyes and it occurs via different pathophysiologic mechanisms. 10 An early postoperative rise in IOP can be due to pupillary block, inflammation, pre-existing glaucoma, and/or final migration of SO into the anterior chamber with consequent mechanical impediment to filtration.…”

Section: Discussionmentioning

confidence: 99%

“…10 Raised IOP remains a significant complication of intraocular SO with reported incidences of between 6 and 40% of eyes and it occurs via different pathophysiologic mechanisms. 10 An early postoperative rise in IOP can be due to pupillary block, inflammation, pre-existing glaucoma, and/or final migration of SO into the anterior chamber with consequent mechanical impediment to filtration. Our case had also raised IOP in early In spite of the great benefits in repairing complicated retinal detachments, long-lasting retinal tamponade of SO have some frequent complications like cataract, glaucoma or keratopathy.…”

Section: Discussionmentioning

confidence: 99%

Göz içi Silikon Yağı Tamponatı ile İlişkili Akut Santral Retinal Arter Tıkanıklığı

...

2012

Tjo

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Sum maryMany systemic and ocular factors may cause acute central retinal artery occlusion (CRAO). Herein, we aimed to describe a case of CRAO due to intraocular silicone oil (SO) tamponade. To the best of our knowledge, a case like our has not been reported previously. A 58-yearold male patient had undergone combined pars plana vitrectomy-lensectomy and intraocular SO for lens luxation and vitreus hemorrhage associated with a blunt ocular trauma in his right eye. Two weeks after the surgery, he presented with acute vision loss in the same eye. He was diagnosed with acute CRAO and it should be related with mechanical press or raised intraocular pressure (IOP) associated with SO. He was treated by partial removal of SO immediately. In spite of the regression of retina edema, his visual acuity did not improve due to optic atrophy. SO may cause CRAO due to raised IOP and/or its mechanical pressure and this complication must be kept in mind. ÖzetPek çok sistemik ve oküler hastalık, akut santral retinal arter tıkanıklığına (SRAT) sebep olmaktadır. Çalışmamızda, daha önce benzer bir hastanın yayınlanmadığını düşündüğümüz, göz içi silikon yağı tamponatı ile ilişkili olarak gelişen SRAT olgusunu sunmayı amaçladık. 58 yaşındaki erkek olgunun sağ gözüne, künt göz travması sonrasında pars plana vitrektomi, lensektomi ve göz içine silikon yağı enjeksiyonu yapıldı. Cerrahiden iki hafta sonra aynı gözde akut görme kaybı gelişti. Olgumuzda, silikon yağının doğrudan basısı ya da yüksek göz içi basıncı (GİB) ile alakalı olduğu düşünülen, akut SRAT saptandı. Acil olarak, göz içinden silikon yağının bir kısmı çıkartıldı. Retinal ödem gerilemesine rağmen, hastanın görme keskinliğinde düzelme olmadı. Silikon yağının, doğrudan bası ve/veya yüksek GİB nedeniyle akut SRAT'na sebep olabileceği akılda tutulmalıdır. (Turk J Oph thal mol 2012; 42: 238-40) Anah tar Ke li me ler: Akut santral retinal arter tıkanıklığı, silikon yağı, pars plana vitrektomi, lensektomi

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“…The secondary glaucoma following the use of SiO with retinal surgery may be caused by migration of emulsified oil to the anterior chamber, synechial angle closure, rubeosis iridis, pupillary block, and inflammation, or a combination of these. 17,18 However, prophylactic surgical inferior iridectomy reduces the risk of pupillary block glaucoma in aphakic eyes. The migration of emulsified oil to the anterior chamber and into the trabecular meshwork may mechanically obstruct the trabecular meshwork or it may cause inflammation of the trabecular meshwork, leading to decreased outflow facility.…”

Section: Discussionmentioning

confidence: 99%

“…[13][14][15][16] Among the risk factors for IOP elevation after SiO injection, a history of preexisting glaucoma, SiO bubbles in the anterior chamber, emulsification of the SiO, diabetes mellitus, and aphakia have been reported. 9,14,17,18 Although the exact mechanism of IOP elevation is unknown, there are many theories about the role of SiO in IOP elevation. The secondary glaucoma following the use of SiO with retinal surgery may be caused by migration of emulsified oil to the anterior chamber, synechial angle closure, rubeosis iridis, pupillary block, and inflammation, or a combination of these.…”

Section: Discussionmentioning

confidence: 99%