1989
DOI: 10.1016/0278-2391(89)90191-2
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Simple bone cyst of the mandibular condyle

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Cited by 27 publications
(14 citation statements)
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“…The local ischemia seems to be due to an arterial dysfunction, along with altered intramedullary venous drainage, which might be interrupted. 15,16 Cyst growth then may be enhanced by the increased intramedullary pressure due to an overflow of intracystic fluid. 17 This fluid, which is very similar to blood serum, contains enzymatic elements from osseous remodeling areas present on the SBC wall, which contains significant vascularity.…”
Section: Etiopathogenesismentioning
confidence: 99%
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“…The local ischemia seems to be due to an arterial dysfunction, along with altered intramedullary venous drainage, which might be interrupted. 15,16 Cyst growth then may be enhanced by the increased intramedullary pressure due to an overflow of intracystic fluid. 17 This fluid, which is very similar to blood serum, contains enzymatic elements from osseous remodeling areas present on the SBC wall, which contains significant vascularity.…”
Section: Etiopathogenesismentioning
confidence: 99%
“…15,25 In this respect, the connection between the prevalence of central giant cell granulomas in the epiphyseal and symphyseal areas and the tumoral degeneration hypothesis remains a matter of debate.…”
Section: Tumor Degenerationmentioning
confidence: 99%
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“…In initial differential diagnosis, benign and malign types of lesions could not be excluded 1,3,5,7,10 . Possible histologies included eosinophilic granuloma, central giant cell granuloma or brown tumour of hyperparathyroidism, ameloblastoma, keratocyst, myxoma, aneurysmal bone cyst and solitary bone cyst 6 ; osteosarcoma and multiple myeloma were possibilities amongst the malignant diseases.…”
Section: Discussionmentioning
confidence: 98%
“…Cette nécrose osseuse, aseptique, permettrait d'expliquer l'origine vasculaire du KOS : il serait secondaire à une ischémie locale, induite par une hémorragie intramédullaire d'origine artérielle qui entraînerait une altération du drainage veineux réalisé par le réseau intramé-dullaire [17,18]. Cette hypothèse semble corroborée par la fréquence élevée des KOS observées chez les sujets atteints de drépanocytose ; cette maladie génétique, à transmission autosomique récessive, qui affecte la synthèse de l'hémoglo-bine, se traduit par des crises douloureuses dues à des microinfarctus, y compris dans l'os, secondaires à des phénomènes vaso-occlusifs [19].…”
Section: Processus Pathogèneunclassified