Sleep-Disordered Breathing and Hormones

Saaresranta, Tarja; Polo, Olli

doi:10.1007/0-387-23692-9_23

Cited by 4 publications

(3 citation statements)

References 237 publications

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“…This neuroendocrine dysfunction is related to the severity of the sleep apnea, as indicated by the nadir levels of arterial oxygen desaturation and the rate of desaturation episodes [39]. The predominance of SAS in males has been attributed to T-mediated aggravation of sleepdisordered breathing (SDB) or to the lack of a protective effect of female hormones [40].…”

Section: The Impact Of Sleep Apnea Syndrome Treatment On Erectile Funmentioning

confidence: 99%

Sleep apnea syndrome and erectile dysfunction

Teloken

Mulhall²

2006

Curr sex health rep

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Erectile dysfunction (ED), defined as the consistent inability to obtain and/or maintain penile erection sufficient for satisfactory sexual relations, is associated with a variety of medical, psychological, and lifestyle risk factors. Estimates for ED incidence in the United States range from 20% to 50%, depending on the epidemiologic study consulted. Sleep apnea syndrome (SAS) affects an estimated 4% of men between the ages of 30 and 60 years. This syndrome is characterized by recurrent episodes of upper airway obstruction during sleep, and it is usually associated with loud snoring and increased daytime sleepiness. It also results in marked sleep fragmentation and decreased rapid eye movement (REM) sleep. This decrease in REM sleep leads to a decrease in nocturnal erectile activity. Due to the loss of protective erections during REM sleep, some researchers have suggested that SAS may be a risk factor for ED. Both ED and SAS affect millions of men. Both diseases have a significant negative impact upon quality of life and interpersonal relationships. Sleep apnea syndrome represents a risk factor for ED, and the severity of both conditions shows a correlation.

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Section: The Impact Of Sleep Apnea Syndrome Treatment On Erectile Funmentioning

confidence: 99%

Sleep apnea syndrome and erectile dysfunction

Teloken

Mulhall²

2006

Curr sex health rep

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“…In patients undergoing tonsillectomy or adenoidectomy, in-lab polysomnography should be repeated 6 weeks to 3 months following surgery [52]. Some children with FD receive growth hormone therapy for short stature [17, 65]. Due to the potential risk of adenoid and tonsil tissue enlargement, in-lab polysomnography should be performed both before, and 6 to 8 weeks after, initiating growth hormone therapy.…”

Section: Recommendations For the Evaluation Of Respiratory Disorders mentioning

confidence: 99%

Respiratory care in familial dysautonomia: Systematic review and expert consensus recommendations

Kazachkov

Palma

Norcliffe‐Kaufmann

et al. 2018

Respiratory Medicine

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Background Familial dysautonomia (Riley-Day syndrome, hereditary sensory autonomic neuropathy type-III) is a rare genetic disease caused by impaired development of sensory and afferent autonomic nerves. As a consequence, patients develop neurogenic dysphagia with frequent aspiration, chronic lung disease, and chemoreflex failure leading to severe sleep disordered breathing. The purpose of these guidelines is to provide recommendations for the diagnosis and treatment of respiratory disorders in familial dysautonomia. Methods We performed a systematic review to summarize the evidence related to our questions. When evidence was not sufficient, we used data from the New York University Familial Dysautonomia Patient Registry, a database containing ongoing prospective comprehensive clinical data from 670 cases. The evidence was summarized and discussed by a multidisciplinary panel of experts. Evidence-based and expert recommendations were then formulated, written, and graded using the Grading of Recommendations, Assessment, Development, and Evaluation (GRADE) system. Results Recommendations were formulated for or against specific diagnostic tests and clinical interventions. Diagnostic tests reviewed included radiological evaluation, dysphagia evaluation, gastroesophageal evaluation, bronchoscopy and bronchoalveolar lavage, pulmonary function tests, laryngoscopy and polysomnography. Clinical interventions and therapies reviewed included prevention and management of aspiration, airway mucus clearance and chest physical therapy, viral respiratory infections, precautions during high altitude or air-flight travel, noninvasive ventilation during sleep, antibiotic therapy, steroid therapy, oxygen therapy, gastrostomy tube placement, Nissen fundoplication surgery, scoliosis surgery, tracheostomy and lung lobectomy. Conclusions Expert recommendations for the diagnosis and management of respiratory disease in patients with familial dysautonomia are provided. Frequent reassessment and updating will be needed.

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“…Several epidemiologic and experimental studies showed that chronic partial sleep loss is associated with increased risk of obesity and obesity-related disorders such as impaired glucose tolerance. The oxygen desaturation index (number of episodes of reduction in oxygen saturation by .4%, per hour of sleep) is a better predictor of insulin resistance than BMI [62]. OSA is associated with prevalent glucose intolerance and insulin resistance even after adjustment for confounders, especially obesity in the general population [60].…”

Section: Sdb and Gestational Diabetes Mellitusmentioning

confidence: 99%

Sleep Disorders and Pregnancy

Ursavaş

2011

Neurological Disorders and Pregnancy

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Sleep-Disordered Breathing and Hormones

Cited by 4 publications

References 237 publications

Sleep apnea syndrome and erectile dysfunction

Sleep apnea syndrome and erectile dysfunction

Respiratory care in familial dysautonomia: Systematic review and expert consensus recommendations

Sleep Disorders and Pregnancy

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