Social Dominance and 22-Year All-Cause Mortality in Men

Bk, Houston; Ma, Babyak; Ma, Chesney; G, Black; Dr, Ragland

doi:10.1097/00006842-199701000-00002

Cited by 103 publications

(27 citation statements)

References 23 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Consistent with these findings, the cardiovascular correlates of trait dominance mentioned above seem to suggest a more adaptive stress-related response for high compared to low dominant subjects (Dienstbier, 1989;Tomaka et al, 1993). This interpretation, however, contradicts empirical findings that found dominant behavior to be related to coronary heart disease (CHD) and allcause mortality (Houston et al, 1992;Houston et al, 1997). Research on nonhuman primates and human subjects indicates that the psychosomatic risk of dominant behavior may partly be determined by environmental characteristics.…”

Section: Introductionmentioning

confidence: 84%

Effects of trait dominance on psychological and cardiovascular responses to social influence attempts: the role of gender and partner dominance

Gramer

Berner

2005

International Journal of Psychophysiology

View full text Add to dashboard Cite

Section: Introductionmentioning

confidence: 84%

Effects of trait dominance on psychological and cardiovascular responses to social influence attempts: the role of gender and partner dominance

Gramer

Berner

2005

International Journal of Psychophysiology

View full text Add to dashboard Cite

“…Other personality types have included "type D" personality, 17 "social dominance," 58 and a "hardy personality" construct 59 ; these latter personality types have not been widely studied as potential risk factors for CAD. Interest in type A behavior accelerated after the Western Collaborative Group Study, which reported that type A behavior was associated with a 2-fold increased risk of CAD and 5-fold increased risk of recurrent MI over an 8.5-year follow-up.…”

Section: Personality and Character Traitsmentioning

confidence: 99%

Impact of Psychological Factors on the Pathogenesis of Cardiovascular Disease and Implications for Therapy

1999

View full text Add to dashboard Cite

Abstract-Recent studies provide clear and convincing evidence that psychosocial factors contribute significantly to the pathogenesis and expression of coronary artery disease (CAD). This evidence is composed largely of data relating CAD risk to 5 specific psychosocial domains: (1) depression, (2) anxiety, (3) personality factors and character traits, (4) social isolation, and (5) chronic life stress. Pathophysiological mechanisms underlying the relationship between these entities and CAD can be divided into behavioral mechanisms, whereby psychosocial conditions contribute to a higher frequency of adverse health behaviors, such as poor diet and smoking, and direct pathophysiological mechanisms, such as neuroendocrine and platelet activation. An extensive body of evidence from animal models (especially the cynomolgus monkey, Macaca fascicularis) reveals that chronic psychosocial stress can lead, probably via a mechanism involving excessive sympathetic nervous system activation, to exacerbation of coronary artery atherosclerosis as well as to transient endothelial dysfunction and even necrosis. Evidence from monkeys also indicates that psychosocial stress reliably induces ovarian dysfunction, hypercortisolemia, and excessive adrenergic activation in premenopausal females, leading to accelerated atherosclerosis.Also reviewed are data relating CAD to acute stress and individual differences in sympathetic nervous system responsivity. New technologies and research from animal models demonstrate that acute stress triggers myocardial ischemia, promotes arrhythmogenesis, stimulates platelet function, and increases blood viscosity through hemoconcentration. In the presence of underlying atherosclerosis (eg, in CAD patients), acute stress also causes coronary vasoconstriction. Recent data indicate that the foregoing effects result, at least in part, from the endothelial dysfunction and injury induced by acute stress. Hyperresponsivity of the sympathetic nervous system, manifested by exaggerated heart rate and blood pressure responses to psychological stimuli, is an intrinsic characteristic among some individuals. Current data link sympathetic nervous system hyperresponsivity to accelerated development of carotid atherosclerosis in human subjects and to exacerbated coronary and carotid atherosclerosis in monkeys.Thus far, intervention trials designed to reduce psychosocial stress have been limited in size and number. Specific suggestions to improve the assessment of behavioral interventions include more complete delineation of the physiological mechanisms by which such interventions might work; increased use of new, more convenient "alternative" end points for behavioral intervention trials; development of specifically targeted behavioral interventions (based on profiling of patient factors); and evaluation of previously developed models of predicting behavioral change. The importance of maximizing the efficacy of behavioral interventions is underscored by the recognition that psychosocial stresses tend to cluster together. ...

show abstract

“…Algunos estudios, sin embargo, han puesto de relieve la existencia de datos respuesta y tasa rápida del habla, Houston et al, 1997), se han asociado a incrementos de la presión sanguínea (Brown y Smith, 1992) o a incrementos por cualquier causa de la mortalidad (Houston et al, 1997). En esta línea de evidencia, Guyll y Contrada (1998) observaron elevaciones de la presión sanguínea entre las personas más hostiles sólo durante los periodos de interacción social.…”

Section: La Hostilidad: ¿Un Factor Gene-ral De Riesgo Cardiovascular?unclassified

Papel de las emociones negativas en el trastono cardiovascular : un análisis crítico

Sandín¹

2002

RPPC

View full text Add to dashboard Cite

En el presente estudio se revisa brevemente la literatura sobre la relación entre las emociones negativas y la enfermedad arterial coronaria (EAC). Numerosos estudios longitudinales y retrospectivos han puesto de relieve que la hostilidad y los síntomas o síndro-mes de ansiedad y depresión están positivamente relacionados con el inicio de la EAC o con la evolución clínica una vez que la EAC se ha producido. La depresión se ha relacionado de forma clara y consistente con la morbilidad y mortalidad asociados a la EAC, con independencia de la gravedad de la enfermedad y otros factores de riesgo. La evidencia sobre el papel de la depresión en el comienzo del la EAC es menos consistente, si bien un componente de la depresión, conocido como «agotamiento vital», podría estar particularmente involucrado. Existe evidencia preliminar y consistente sobre la implicación de la ansiedad en el inicio y evolución clínica de la EAC. Los datos sobre una posible asociación entre la hostilidad y el inicio de la EAC son limitados pero sugestivos. No existe evidencia fiable sobre la relación entre la hostilidad y el curso (evolución clínica) de la EAC. Existe información adicional que sugiere una estrecha relación entre la depresión (y/o ansiedad) y la calidad de vida en los pacientes que han sufrido infarto de miocardio. La no consideración de las relaciones que pueden darse entre los tres tipos de emociones podría explicar algunas de las inconsistencias que se han encontrado en la investigación sobre las emociones negativas y el trastorno cardiovascular.Palabras clave: ansiedad, depresión, hostilidad, emoción negativa, enfermedad arterial coronaria, infarto de miocardio, estrés. Negative emotions in cardiovascular disorder: A critical analysisThis study briefly reviews the literature on relationship between negative emotions and coronary heart disease (CHD). Numerous longitudinal and retrospective studies have found that hostility and symptoms or syndromes of anxiety and depression are positively related to the onset of CHD or to outcomes after CHD is manifest. Depression has been shown to be clearly related to morbidity and mortality after CHD, independent of disease severity and other risk factors. Evidence for a role of depression in the onset of CHD is quite mixed, although the «vital exhaustion» component could be relevant. Evidence that anxiety is involved in the onset of CHD as well as in outcomes after CHD is preliminary but strong. Evidence for an association between hostility and CHD is limited but suggestive. Hostility is not reliably related to morbidity or mortality after CHD. In addition, depression (and/or anxiety) after myocardial infarction appear to be powerfully related to later overall quality of life among survivors. Disregard of relationships between the three emotions may explain some unresolved issues and nuil findings in the research on negative emotions and cardiovascular disorder.

show abstract

Social Dominance and 22-Year All-Cause Mortality in Men

Abstract: These results suggest that, in future research concerning psychosocial factors and long-term survival, attention should be given to social dominance as well as to hostility.

Cited by 103 publications

References 23 publications

Effects of trait dominance on psychological and cardiovascular responses to social influence attempts: the role of gender and partner dominance

Effects of trait dominance on psychological and cardiovascular responses to social influence attempts: the role of gender and partner dominance

Impact of Psychological Factors on the Pathogenesis of Cardiovascular Disease and Implications for Therapy

Papel de las emociones negativas en el trastono cardiovascular : un análisis crítico

Contact Info

Product

Resources

About