BackgroundLoin/flank pain in pregnancy is a common presentation to the Emergency Department (ED). There are a wide range of causes: medical, surgical, urological, obstetrical and gynaecological, but the predominant cause is acute hydronephrosis [1]. Hydroureteronephrosis is frequently associated with pregnancy and on rare occasions can progress to rupture of the urinary tract. This paper describes the case of a young female who developed an initial rupture of the left ureter, progressing following admission to develop symptoms on the right side. Also we have included a narrative review of the literature on this subject. To date, we believe, this is the first case report of a patient with bilateral symptomatic upper urinary tract hydroureteronephrosis.
Case PresentationA 31 year old white European primigravida (28 weeks gestational age) with an uncomplicated pregnancy presented to the Emergency Department (ED) complaining of severe progressive left flank pain. The symptoms had commenced approximately 2 hours prior, whilst travelling by train, with gradual onset of colic pain of her left groin, worsening and ascending to the left flank and renal angle. Her past medical history included an episode of left pyelonephritis four years previous, bilateral breasts reduction two years ago and mild eczema which improved with progression of her pregnancy. She had no allergies and was not on any current medication. There was no family history of significance. On examination she was alert and oriented (GCS 15/15), not pale or cyanotic, anxious, restless, and complaining of severe pain (10/10 on numeric pain rating scale). Her body temperature was normal (36.6°C), with mild tachycardia (110 bpm), BP 140/105 mmHg, respiratory rate 17 breaths/minute, SaO 2 96% on room air and a capillary refill time < 2 sec. Percussion tenderness was elicited of the left loin. Her uterus had a normal height for the gestational age; there was no vaginal discharge or bleeding. Foetal movements were present with a heart rate of 120 bpm. The remainder of the clinical exam was unremarkable. An 18 G Intravenous (IV) cannula was inserted and baseline bloods investigations drawn for full blood count, group and screen, urea and creatinine, electrolytes, glucose, liver function test's, "C" Reactive Protein (CRP) and Venous Blood Gas (VBG). She was initially managed with opioid analgesia (pethidine), intravenous paracetamol and maintenance fluids (0.9% NaCl). Shortly after her pain score subsided to 3-4/10.No free fluid or other abnormalities were revealed by an ED Ultrasound (US). A bed side urinalysis revealed a moderate amount of blood, but negative for nitrites and leukocytes. Her VBG and glucose were normal.Based on the clinical presentation and microscopic haematuria a provisional diagnosis of renal colic secondary to a possible migrant renal calculus was established.Laboratory blood results revealed an elevated WBC (14.5×10 9 /L) with mild neutrophlia (11.8×10 9 /L) and elevated CRP (8.3 mg/L). Urea, creatinine, LFT's and electrolytes were normal. P...