Spontaneous rupture of the stomach: A complication of bulimia

Breslow, Michael J.; Yates, Alayne; Shisslak, Catherine M.

doi:10.1002/1098-108x(198601)5:1<137::aid-eat2260050113>3.0.co;2-v

Cited by 22 publications

(20 citation statements)

References 14 publications

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“…Another sign reported in the literature is the inability to vomit which is not fully understood. This may be due to the occlusion of the gastroesophageal junction by the distended fundus, which angulates the esophagus against the right crus of the diaphragm, producing a one-way valve [20]. Significant, diffuse abdominal distension accompanied by abdominal pain is common.…”

Section: Discussionmentioning

confidence: 99%

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Sahoo

Kumar

Jaiswal

et al. 2013

Case Reports in Surgery

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Acute gastric dilatation can have multiple etiologies which may lead to ischemia of the stomach. Without proper timely diagnosis and treatment, potentially fatal events such as gastric perforation, haemorrhage, and other serious complications can occur. Here we present a 36-year-old man who came to the casualty with pain abdomen and distension for 2 days. Clinically, abdomen was asymmetrically distended more in the left hypochondrium and epigastrium region. Straight X-ray abdomen showed opacified left hypochondrium with nonspecific gaseous distension of bowel. Exploratory laparotomy revealed dilated stomach with patchy gangrene over lesser curvature and fundic area. About 4 litres of brownish fluid along with semisolid undigested food particles was sucked out (mainly undigested pieces of meat). Limited resection of gangrenous areas and primary repair were done along with feeding jejunostomy. Necrosis of the stomach was confirmed on histopathology. The patient recovered well and was discharged on the tenth postoperative day.

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Section: Discussionmentioning

confidence: 99%

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Sahoo

Kumar

Jaiswal

et al. 2013

Case Reports in Surgery

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“… 63 Acute gastric dilitation and rupture is rare but is associated with a mortality rate of 80% when it occurs. 64–66 Other gastrointestinal complications include postbinge pancreatitis and constipation due to laxative abuse. 67 Cathartic colon with toxic degeneration of Auerbach's plexus due to overuse of stimulant laxatives can render the colon unable to perform normal peristalsis without large doses of laxatives.…”

Section: Treatment Issuesmentioning

confidence: 99%

Detection, evaluation, and treatment of eating disorders

2000

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OBJECTIVE:To describe how primary care clinicians can detect an eating disorder and identify and manage the associated medical complications. DESIGN:A review of literature from 1994 to 1999 identified by a MEDLINE search on epidemiology, diagnosis, and therapy of eating disorders, including anorexia nervosa and bulimia nervosa. MEASUREMENTS AND MAIN RESULTS:Detection requires awareness of risk factors for, and symptoms and signs of, anorexia nervosa (e.g., participation in activities valuing thinness, family history of an eating disorder, amenorrhea, lanugo hair) and bulimia nervosa (e.g., unsuccessful attempts at weight loss, history of childhood sexual abuse, family history of depression, erosion of tooth enamel from vomiting, partoid gland swelling, and gastroesophageal reflux). Providers must also remain alert for disordered eating in female athletes (the female athlete triad) and disordered eating in diabetics. Treatment requires a multidisciplinary team including a primary care practitioner, nutritionist, and mental health professional. The role of the primary care practitioner is to help determine the need for hospitalization and to manage medical complications (e.g., arrhythmias, refeeding syndrome, osteoporosis, and electrolyte abnormalities such as hypokalemia). E ating disorders are common in young women. Although the evaluation and treatment of eating disorders has typically been thought to be the realm of the psychiatrist, the primary care physician plays an important role. In the era of managed care, the primary care physician may be the first to detect an eating disorder and is responsible for coordinating care, including management of complications and determining the need for hospitalization. In addition, a patient may need to see a primary care physician first in order to be referred to a mental health clinician. The primary care physician must work with a multidisciplinary team, including a nutritionist and a mental health specialist to provide well-integrated care for patients with eating disorders. Finally, particularly for those patients who have milder forms of disordered eating and may not be seeing a mental health specialist regularly, the primary care physician may have primary responsibility for ongoing care and for management of complications. The following questions will be addressed in this review: What signs and symptoms should lead the primary care physician to suspect an eating disorder? Are there high risk groups at particular risk for eating disorders? What laboratory tests should be part of the evaluation of a patient with a suspected eating disorder? What are common medical complications among patients with eating disorders? What are the data on current treatments for patients with eating disorders? CONCLUSION: METHODSThree authors did a MEDLINE search on epidemiology, diagnosis, and therapy of eating disorders, anorexia nervosa, bulimia nervosa, and disordered eating. Search terms included anorexia nervosa, bulimia nervosa, disordered eating, and female athlete triad. Other ...

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“…The situation resolved when the patient was eventually able to regurgitate the stomach contents. A 30-year-old woman ingested a large volume of food and made several unsuccessful attempts to regurgitate (68). Sodium bicarbonate was ingested but did not initiate vomiting.…”

Section: Stomachmentioning

confidence: 99%

Physiological Effects of Bulimia Nervosa on the Gastrointestinal Tract

Anderson¹,

Shaw²,

McCargar³

1997

Canadian Journal of Gastroenterology

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Bulimia nervosa is an eating disorder characterized by frequent bouts of binge eating accompanied by compensatory behaviour for preventing weight gain (purging). It is estimated that 3% to 5% of young women are affected by bulimia nervosa, and its prevalence is increasing. Bulimia nervosa afflicts both sexes and all races. It can lead to serious medical complications. The expression of the disease in the gastrointestinal tract may have a critical role in the diagnosis of bulimia nervosa. Physiological effects of bulimia nervosa on the gastrointestinal tract include dental caries and enamel erosion; enlargement of the parotid gland; esophagitis; changes in gastric capacity and gastric emptying; gastric necrosis; and alterations of the intestinal mucosa. Identification of any of these factors may aid in establishing an early diagnosis, which has been shown to increase the likelihood of recovery.

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Spontaneous rupture of the stomach: A complication of bulimia

Cited by 22 publications

References 14 publications

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Detection, evaluation, and treatment of eating disorders

Physiological Effects of Bulimia Nervosa on the Gastrointestinal Tract

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